Recovery of higher-order cognitive deficits after brain hypoxia or frontomedial vascular lesions.
In accordance with neurobiological data, thought impairment after brain hypoxia was mainly (though not exclusively) attributed to memory and attentional dysfunction. As opposed to this, problem-solving defects due to frontomedial lesions seem to be determined essentially by such behavioral alterations such as apathy, distractibility and reduced insight. Creative and adaptive problem solving abilities could not be restored in both groups, at least not within the first year post onset.
von Cramon
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DY
Max-Planck-Institute for Cognitive Neuroscience, Leipzig, Germany.
Matthes-von Cramon
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