title: [Stereomorphometric study of target organs in rats with hereditary stress-induced arterial hypertension at different periods of postnatal ontogenesis under changed conditions of nursing].
abstract: In rats with inherited stress-induced arterial hypertension (ISIAH strain), myocardium, adrenal gland, and renal glomerular apparatus were studied at different periods of postnatal ontogenesis (3 weeks and 6 months) to assess the influence of the changed conditions of nursing on the development of a hypertensive status and structural-functional characteristics of target organs. It was demonstrated that nursing of rats by foster normotensive Wistar females exerted a modulating influence upon the realization of stress-determined program of arterial hypertension development, seemingly delaying it and alleviating the negative consequences in respect to the target organs, through increased effectiveness of recruitment of adaptive-compensatory organism reserves, however, on the whole, it did not interrupt this program.
authors.forename: M D
authors.initials: MD
authors.lastname: Shmerling
authors.forename: I I
authors.initials: II
authors.lastname: Buzueva
authors.forename: I M
authors.initials: IM
authors.lastname: Korostyshevskaia
authors.forename: V A
authors.initials: VA
authors.lastname: Lazarev
authors.forename: V F
authors.initials: VF
authors.lastname: Maksimov
authors.forename: E E
authors.initials: EE
authors.lastname: Filiushina
authors.forename: A L
authors.initials: AL
authors.lastname: Markel'
authors.forename: G S
authors.initials: GS
authors.lastname: Iakobson
mesh_terms: D000311:Adrenal Glands / Q000254:growth & development / Q000648:ultrastructure*; D000818:Animals; D000833:Animals, Suckling; D005247:Feeding Behavior; D005260:Female; D006321:Heart / Q000254:growth & development; D006973:Hypertension / Q000209:etiology* / Q000235:genetics; D007678:Kidney Glomerulus / Q000254:growth & development / Q000648:ultrastructure*; D009206:Myocardium / Q000648:ultrastructure*; D051381:Rats; D017208:Rats, Wistar; D013312:Stress, Physiological / Q000150:complications* / Q000235:genetics