Basic mechanisms in status epilepticus: role of calcium in neuronal injury and the induction of epileptogenesis.
DeLorenzo
Robert J
RJ
Department of Neurology, Pharmacology, and Toxicology and Biochemistry and Molecular Biophysics, Virginia Commonwealth University School of Medicine, Richmond, Virginia, USA.
Sun
David A
DA
D000818:Animals; D002118:Calcium / Q000378:metabolism*; D016923:Cell Death / Q000187:drug effects / Q000502:physiology; D004347:Drug Interactions; D006801:Humans; D007608:Kainic Acid; D009474:Neurons / Q000187:drug effects / Q000473:pathology*; D010862:Pilocarpine; D016194:Receptors, N-Methyl-D-Aspartate / Q000502:physiology; D013226:Status Epilepticus / Q000139:chemically induced / Q000378:metabolism* / Q000503:physiopathology*; D013997:Time Factors; D005680:gamma-Aminobutyric Acid / Q000378:metabolism
D016194:Receptors, N-Methyl-D-Aspartate; D010862:Pilocarpine; D005680:gamma-Aminobutyric Acid; D007608:Kainic Acid; D002118:Calcium