A minimal set of DNA repair genes is sufficient for survival of Coxiella burnetii under oxidative stress.
This paper studies the functional and regulatory role of RecA, but further investigations are necessary to clarify the control mechanism of the SOS response gene expression in C. burnetii.
Department of Medical Microbiology and Immunology, Texas A&M University System Health Science Center, College Station, TX 77843-1114, USA.
K
K
Mertens
L
L
Lantsheer
J E
JE
Samuel
D016997:Coxiella burnetii / Q000201:enzymology / Q000235:genetics* / Q000254:growth & development* / Q000378:metabolism; D004260:DNA Repair / Q000235:genetics*; D004269:DNA, Bacterial / Q000235:genetics*; D004926:Escherichia coli / Q000235:genetics; D018384:Oxidative Stress / Q000235:genetics*; D011938:Rec A Recombinases / Q000235:genetics / Q000378:metabolism
D004269:DNA, Bacterial; D011938:Rec A Recombinases