How senescent vascular cells lose their Clock age-dependent impairment of circadian rhythmicity in smooth muscle cells.
Illi
Barbara
B
Gaetano
Carlo
C
Capogrossi
Maurizio C
MC
D056930:ARNTL Transcription Factors; D000818:Animals; D051792:Basic Helix-Loop-Helix Proteins / Q000235:genetics; D056926:CLOCK Proteins; D018797:Cell Cycle Proteins; D016922:Cellular Senescence; D002940:Circadian Rhythm; D017362:Cyclic AMP Response Element-Binding Protein / Q000502:physiology; D005786:Gene Expression Regulation; D006801:Humans; D009131:Muscle, Smooth, Vascular / Q000166:cytology / Q000378:metabolism*; D032389:Myocytes, Smooth Muscle / Q000166:cytology / Q000378:metabolism*; D009569:Nitric Oxide / Q000502:physiology; D009687:Nuclear Proteins / Q000235:genetics; D056950:Period Circadian Proteins; D015534:Trans-Activators / Q000235:genetics*
D056930:ARNTL Transcription Factors; D051792:Basic Helix-Loop-Helix Proteins; D056926:CLOCK Proteins; D018797:Cell Cycle Proteins; D017362:Cyclic AMP Response Element-Binding Protein; D009569:Nitric Oxide; D009687:Nuclear Proteins; D056950:Period Circadian Proteins; D015534:Trans-Activators; C538960:BMAL1 protein, human; C539022:PER1 protein, human; C538943:CLOCK protein, human