[Acute tubular necrosis: Pathogenesis, pathophysiology, and therapy].
Yonemura
Katsuhiko
K
Department of Internal Medicine, Fujinomiya City General Hospital.
Goto
Tetsuo
T
Sakao
Yukitoshi
Y
Endo
Mikiko
M
D020170:Caspase 1 / Q000502:physiology; D002455:Cell Division / Q000235:genetics; D018932:Chemokine CCL2 / Q000502:physiology; D017228:Hepatocyte Growth Factor / Q000502:physiology / Q000627:therapeutic use; D006801:Humans; D018799:Intercellular Adhesion Molecule-1 / Q000502:physiology; D007683:Kidney Tubular Necrosis, Acute / Q000145:classification / Q000209:etiology / Q000628:therapy; D007684:Kidney Tubules / Q000166:cytology / Q000502:physiology; D011065:Poly(ADP-ribose) Polymerases / Q000502:physiology; D017382:Reactive Oxygen Species; D012038:Regeneration / Q000235:genetics; D048069:Tumor Necrosis Factors / Q000502:physiology
C111835:CCL2 protein, human; D018932:Chemokine CCL2; D017382:Reactive Oxygen Species; D048069:Tumor Necrosis Factors; D018799:Intercellular Adhesion Molecule-1; D017228:Hepatocyte Growth Factor; D011065:Poly(ADP-ribose) Polymerases; D020170:Caspase 1