Decrease in anterior knee laxity by electrical stimulation of normal and reconstructed anterior cruciate ligaments.
We have investigated the changes in anterior laxity of the knee in response to direct electrical stimulation of eight normal and 45 reconstructed anterior cruciate ligaments (ACLs). In the latter, the mean time from reconstruction was 26.7 months (24 to 32). The ACL was stimulated electrically using a bipolar electrode probe during arthroscopy. Anterior laxity was examined with the knee flexed at 20 degrees under a force of 134 N applied anteriorly to the tibia using the KT-2000 knee arthrometer before, during and after electrical stimulation. Anterior tibial translation in eight normal and 17 ACL-reconstructed knees was significantly decreased during stimulation, compared with that before stimulation. In 28 knees with reconstruction of the ACL, in 22 of which the grafts were found to have detectable somatosensory evoked potentials during stimulation, anterior tibial translation was not decreased. These findings suggest that the ACL-hamstring reflex arc in normal knees may contribute to the functional stability and that this may not be fully restored after some reconstructions of the ACL.
Department of Orthopaedic Surgery, Shimane University School of Medicine, Izumo, Shimane 693-8501, Japan. j-iwasa@med.shimane-u.ac.jp
J
J
Iwasa
M
M
Ochi
Y
Y
Uchio
N
N
Adachi
K
K
Kawasaki
D000293:Adolescent; D000328:Adult; D016118:Anterior Cruciate Ligament / Q000503:physiopathology* / Q000601:surgery; D001182:Arthroscopy; D004599:Electric Stimulation Therapy / Q000379:methods*; D005073:Evoked Potentials, Somatosensory / Q000502:physiology; D005260:Female; D006801:Humans; D007593:Joint Instability / Q000503:physiopathology* / Q000628:therapy; D007719:Knee Joint / Q000503:physiopathology* / Q000601:surgery; D008297:Male; D018482:Muscle, Skeletal / Q000503:physiopathology; D009412:Nerve Fibers / Q000502:physiology; D011434:Proprioception / Q000502:physiology; D012026:Reflex, Stretch / Q000502:physiology; D013977:Tibia / Q000503:physiopathology