[ATP1A2: a key player in familial hemiplegic migraine].
Blostein
Rhoda
R
Segall
Laura
L
Gargus
J Jay
JJ
D019943:Amino Acid Substitution; D000818:Animals; D001921:Brain / Q000201:enzymology; D006429:Hemiplegia / Q000235:genetics*; D006801:Humans; D017136:Ion Transport / Q000235:genetics; D007527:Isoenzymes / Q000737:chemistry / Q000235:genetics / Q000502:physiology; D008322:Mammals / Q000378:metabolism; D051379:Mice; D018345:Mice, Knockout; D020325:Migraine with Aura / Q000145:classification / Q000235:genetics*; D008958:Models, Molecular; D020125:Mutation, Missense; D009419:Nerve Tissue Proteins / Q000737:chemistry / Q000235:genetics / Q000502:physiology; D017354:Point Mutation; D011487:Protein Conformation; D000254:Sodium-Potassium-Exchanging ATPase / Q000737:chemistry / Q000235:genetics / Q000502:physiology*
D007527:Isoenzymes; D009419:Nerve Tissue Proteins; C498531:ATP1A2 protein, human; D000254:Sodium-Potassium-Exchanging ATPase