title: Hemodynamic physiology in aortic arch and trigger factors of atherosclerosis.
abstract: Magnetic resonance angiography showed that blood flow in the aortic arch during protodiastole is separated into the opposite flows, which are temporarily arrested at the certain sites creating the flat front of the flow. The protodiastolic antegrade peak acceleration 6-fold surpasses the systolic one. Due to adhesion, the high gradient pressure forces can damage the vascular wall.
authors.affiliation: Department of Magnetic Resonance Tomography, Institute of Medical Radiology, Tbilisi.
authors.forename: F I
authors.initials: FI
authors.lastname: Todua
authors.forename: M V
authors.initials: MV
authors.lastname: Beraya
mesh_terms: D000328:Adult; D001011:Aorta / Q000473:pathology; D001013:Aorta, Thoracic / Q000033:anatomy & histology* / Q000473:pathology*; D050197:Atherosclerosis / Q000473:pathology*; D055592:Biophysical Phenomena; D001703:Biophysics; D001783:Blood Flow Velocity; D003971:Diastole; D004730:Endothelium, Vascular / Q000378:metabolism; D006439:Hemodynamics; D006801:Humans; D018810:Magnetic Resonance Angiography / Q000379:methods; D008297:Male; D013997:Time Factors; D000267:Tissue Adhesions