Withdrawal of vasoconstrictor influences in local metabolic coronary vasodilation.
Tune
Johnathan D
JD
D000818:Animals; D001685:Biological Factors / Q000378:metabolism; D003326:Coronary Circulation / Q000502:physiology*; D003331:Coronary Vessels / Q000187:drug effects / Q000502:physiology; D016861:Cyclooxygenase Inhibitors / Q000008:administration & dosage / Q000494:pharmacology; D016232:Endothelins / Q000494:pharmacology; D005080:Exercise Test; D008833:Microcirculation / Q000502:physiology; D008955:Models, Cardiovascular; D009569:Nitric Oxide / Q000096:biosynthesis; D010101:Oxygen Consumption / Q000187:drug effects* / Q000502:physiology; D010805:Physical Conditioning, Animal; D005082:Physical Exertion; D011453:Prostaglandins / Q000096:biosynthesis; D034421:Sus scrofa; D014661:Vasoconstriction / Q000187:drug effects* / Q000502:physiology; D014662:Vasoconstrictor Agents / Q000494:pharmacology*; D014664:Vasodilation / Q000502:physiology*
D001685:Biological Factors; D016861:Cyclooxygenase Inhibitors; D016232:Endothelins; D011453:Prostaglandins; D014662:Vasoconstrictor Agents; C067410:endothelium-dependent hyperpolarization factor; D009569:Nitric Oxide