HDL--associated paraoxonase 1 (PON1) and dietary antioxidants attenuate lipoprotein oxidation, macrophage foam cells formation and atherosclerosis development.
Aviram
Michael
M
The Lipid Research Laboratory, Technion Faculty of Medicine, The Rappaport Family Institute for Research in the Medical Sciencesand Rambam Medical Center, Haifa, Israel.
D000818:Animals; D000975:Antioxidants / Q000494:pharmacology*; D043303:Aryldialkylphosphatase / Q000378:metabolism / Q000502:physiology*; D050197:Atherosclerosis / Q000209:etiology*; D005487:Foam Cells / Q000166:cytology*; D006801:Humans; D015227:Lipid Peroxidation; D008074:Lipoproteins / Q000378:metabolism; D008075:Lipoproteins, HDL / Q000378:metabolism; D051379:Mice; D009747:Nutritional Physiological Phenomena
D000975:Antioxidants; D008074:Lipoproteins; D008075:Lipoproteins, HDL; D043303:Aryldialkylphosphatase; C468406:PON1 protein, human