[Apoptosis of vascular cells].
Sata
Masataka
M
Department of Cardiovascular Medicine, University of Tokyo Graduate School of Medicine.
D000804:Angiotensin II / Q000502:physiology; D000975:Antioxidants / Q000494:pharmacology; D017209:Apoptosis / Q000187:drug effects / Q000235:genetics / Q000502:physiology; D001161:Arteriosclerosis / Q000209:etiology / Q000473:pathology; D004730:Endothelium, Vascular / Q000166:cytology*; D004967:Estrogens / Q000494:pharmacology / Q000502:physiology; D006133:Growth Substances / Q000502:physiology; D006801:Humans; D006973:Hypertension / Q000209:etiology / Q000473:pathology*; D008070:Lipopolysaccharides; D008077:Lipoproteins, LDL / Q000502:physiology; D009131:Muscle, Smooth, Vascular / Q000166:cytology*; D009569:Nitric Oxide / Q000502:physiology; D018384:Oxidative Stress / Q000502:physiology; D013314:Stress, Mechanical; D014409:Tumor Necrosis Factor-alpha / Q000502:physiology; D019014:fas Receptor / Q000502:physiology
D000975:Antioxidants; D004967:Estrogens; D006133:Growth Substances; D008070:Lipopolysaccharides; D008077:Lipoproteins, LDL; D014409:Tumor Necrosis Factor-alpha; D019014:fas Receptor; C099252:oxidized low density lipoprotein; D000804:Angiotensin II; D009569:Nitric Oxide