Immunology. When F-actin becomes too much of a good thing.
Dustin
Michael L
ML
Progam in Molecular Pathogenesis, Skirball Institute, New York University Medical Center, 540 First Avenue, New York, NY 10016, USA. dustin@saturn.med.nyu.edu
D051376:Actin-Related Protein 2-3 Complex / Q000037:antagonists & inhibitors / Q000378:metabolism; D000199:Actins / Q000378:metabolism*; D000818:Animals; D017209:Apoptosis; D001665:Binding Sites; D016923:Cell Death; D002465:Cell Movement; D002634:Chemotaxis, Leukocyte; D006706:Homeostasis; D007425:Intracellular Membranes / Q000502:physiology; D008564:Membrane Potentials; D051379:Mice; D008840:Microfilament Proteins / Q000737:chemistry / Q000502:physiology*; D008928:Mitochondria / Q000502:physiology*; D017434:Protein Structure, Tertiary; D011948:Receptors, Antigen, T-Cell / Q000276:immunology; D013601:T-Lymphocytes / Q000276:immunology / Q000502:physiology*; D050994:Voltage-Dependent Anion Channels / Q000378:metabolism
D051376:Actin-Related Protein 2-3 Complex; D000199:Actins; D008840:Microfilament Proteins; D011948:Receptors, Antigen, T-Cell; D050994:Voltage-Dependent Anion Channels; C072061:coronin proteins