title: [Coronary embolism with apical ballooning complicating electrical cardioversion--is it part of the apical ballooning syndrome? Case report and review of the literature].
abstract: METHODS
  A case of thromboembolic left anterior descending artery occlusion following electrical cardioversion for atrial fibrillation is described. A 66-year-old female patient presenting with exertional angina pectoris and atrial fibrillation was subjected to coronary angiography, ventriculography and transesophageal echocardiography. No significant coronary stenoses were found, left ventricular systolic function and regional wall motion were normal, and she had no intracardiac thrombi. Direct-current cardioversion was complicated by asystole which was managed by cardiac massage and 1 mg atropine and 1 mg adrenaline intravenously. Shortly afterwards, the patient regained a normal sinus rhythm. She remained hypotensive and developed ST segment elevation over the chest leads. Angiography was repeated and showed apical ballooning and thromboembolic subtotal occlusion of the proximal LAD, which migrated to the periphery and subsequently disappeared with regain of TIMI 3 flow by the end of angiography. 4 months later, a normal left ventricular global and regional function was seen in echocardiography.
  
  
  OBJECTIVE
  At least part of the apical ballooning syndrome patients are a sequel of transient thromboembolic occlusions.
  
  
  METHODS
  Therefore, all patients with the diagnosis of coronary embolism in the period from September 2004 to September 2005 were analyzed retrospectively.
  
  
  RESULTS
  Further three patients had coronary artery embolism (two females, one male; age 69-76 years). Two patients had apical ballooning, and one showed global hypokinesia (known dilated cardiomyopathy). Cardiac markers were slightly elevated. ST segment elevation was seen in two patients and T-wave inversion in one. All had risk factors for embolization and two had an additional triggering factor. Both cases with apical ballooning had regained a normal ejection fraction at follow-up.
  
  
  CONCLUSIONS
  This case series probably bridges the gap between two as yet separate disease entities, namely the apical ballooning syndrome and coronary emboli. The time factor probably plays the pivotal role in determining whether the apical ballooning alone or also an embolus is seen. It seems possible that some patients presenting with apical ballooning are unrecognized coronary thromboembolic cases.
authors.affiliation: Herz-Kreislauf-Zentrum, Segeberger Kliniken GmbH, Akademisches Lehrkrankenhaus der Universität Kiel, Bad Segeberg, Germany. bjoern.buchter@gmx.de
authors.forename: Björn
authors.initials: B
authors.lastname: Buchter
authors.forename: Ahmed A
authors.initials: AA
authors.lastname: Khattab
authors.forename: Gert
authors.initials: G
authors.lastname: Richardt
mesh_terms: D000077284:Abciximab; D000368:Aged; D000792:Angiography; D000911:Antibodies, Monoclonal / Q000008:administration & dosage; D001281:Atrial Fibrillation / Q000628:therapy*; D009202:Cardiomyopathies / Q000175:diagnosis / Q000188:drug therapy / Q000209:etiology*; D017023:Coronary Angiography; D003328:Coronary Thrombosis / Q000175:diagnosis / Q000188:drug therapy / Q000209:etiology*; D003402:Creatine Kinase / Q000097:blood; D004554:Electric Countershock / Q000009:adverse effects*; D005260:Female; D006801:Humans; D007140:Immunoglobulin Fab Fragments / Q000008:administration & dosage; D009200:Myocardial Contraction / Q000187:drug effects / Q000502:physiology*; D013577:Syndrome; D013923:Thromboembolism / Q000175:diagnosis / Q000188:drug therapy / Q000209:etiology*; D018487:Ventricular Dysfunction, Left / Q000175:diagnosis / Q000188:drug therapy / Q000209:etiology*
chemical_list: D000911:Antibodies, Monoclonal; D007140:Immunoglobulin Fab Fragments; D003402:Creatine Kinase; D000077284:Abciximab