[Facial chorea and hemichorea due to cardiogenic cerebral embolism in the cortex and subcortical white matter].
A 62-year-old man was admitted to our hospital because of suddenly developed bilateral facial chorea and left-sided hemichorea. He had neither hemiparesis nor sensory disturbance. Diffusion-weighted magnetic resonance images of the brain showed acute cortical and subcortical infarctions at the right insula, frontal, temporal and parietal lobes. Tiapride hydrochloride was administered for his chorea. The chorea disappeared on the next day. We diagnosed him as cardiogenic cerebral embolism because he had a paroxysmal atrial fibrillation. We supposed that his chorea was induced by interruption of excitatory output from cerebral cortex to striatum and subthalamic nucleus. Contralateral cortical and subcortical infarction must be considered when a patient suddenly develops hemichorea.
Sugiura
Akira
A
Department of Neurology, Iwata City General Hospital.
Fujimoto
Masaya
M
D000208:Acute Disease; D018726:Anti-Dyskinesia Agents / Q000627:therapeutic use; D001281:Atrial Fibrillation / Q000150:complications*; D002540:Cerebral Cortex; D002544:Cerebral Infarction / Q000175:diagnosis / Q000209:etiology*; D002819:Chorea / Q000188:drug therapy / Q000209:etiology*; D005145:Face; D006801:Humans; D020766:Intracranial Embolism / Q000175:diagnosis / Q000209:etiology*; D008279:Magnetic Resonance Imaging; D008297:Male; D008875:Middle Aged; D013976:Tiapamil Hydrochloride / Q000627:therapeutic use
D018726:Anti-Dyskinesia Agents; D013976:Tiapamil Hydrochloride