title: The low incidence of bacterial infections could be a protective factor against variceal bleeding per se in hemodynamic responders to propranolol.
authors.forename: Marco
authors.initials: M
authors.lastname: Senzolo
authors.forename: Evangelos
authors.initials: E
authors.lastname: Cholongitas
authors.forename: Laura
authors.initials: L
authors.lastname: Marelli
authors.forename: Ulrich
authors.initials: U
authors.lastname: Thalheimer
authors.forename: David
authors.initials: D
authors.lastname: Patch
authors.forename: Andrew K
authors.initials: AK
authors.lastname: Burroughs
mesh_terms: D000959:Antihypertensive Agents / Q000627:therapeutic use*; D001424:Bacterial Infections / Q000453:epidemiology*; D004932:Esophageal and Gastric Varices / Q000453:epidemiology*; D006471:Gastrointestinal Hemorrhage / Q000453:epidemiology*; D006801:Humans; D006975:Hypertension, Portal / Q000150:complications / Q000188:drug therapy*; D015994:Incidence; D008103:Liver Cirrhosis / Q000150:complications; D011433:Propranolol / Q000627:therapeutic use*
chemical_list: D000959:Antihypertensive Agents; D011433:Propranolol