Finite element analysis of the spondylolysis in lumbar spine.
Spondylolysis is a fracture of the bone lamina in the pars interarticularis and has a high risk of developing spondylolisthesis, as well as traction on the spinal cord and nerve root, leading to spinal disorders or low back pain when the lumbar spine is subjected to high external forces. Previous studies mostly investigated the mechanical changes of the endplate in spondylolysis. However, little attention has been focused on the entire structural changes that occur in spondylolysis. Therefore, the purpose of this study was to evaluate the biomechanical changes in posterior ligaments, disc, endplate, and pars interarticularis between the intact lumbar spine and spondylolysis. A total of three finite element models, namely the intact L2-L4 lumbar spine, lumbar spine with unilateral pars defect and with bilateral pars defect were established using a software ANSYS 6.0. A loading of 10 N.m in flexion, extension, left torsion, right torsion, left lateral bending, and right lateral bending respectively were imposed on the superior surface of the L2 body. The bottom of the L4 vertebral body was completely constrained. The finite element models estimated that the lumbar spine with a unilateral pars defect was able to maintain spinal stability as the intact lumbar spine, but the contralateral pars experienced greater stress. For the lumbar spine with a bilateral pars defect, the rotation angle, the vertebral body displacement, the disc stress, and the endplate stress, was increased more when compared to the intact lumbar spine under extension or torsion.
Wang
Jung-Pin
JP
Department of Orthopaedic Surgery, Mackay Memorial Hospital, Taipei, Taiwan.
Zhong
Zheng-Cheng
ZC
Cheng
Cheng-Kung
CK
Chen
Chen-Sheng
CS
Yu
Chung-hung
CH
Chang
Ting-Kuo
TK
Wei
Shun-Hwa
SH
D000328:Adult; D019245:Compressive Strength; D003198:Computer Simulation; D004548:Elasticity; D020342:Finite Element Analysis; D006801:Humans; D007593:Joint Instability / Q000209:etiology / Q000503:physiopathology*; D008159:Lumbar Vertebrae / Q000503:physiopathology*; D008297:Male; D008954:Models, Biological; D016059:Range of Motion, Articular; D013169:Spondylolysis / Q000150:complications / Q000503:physiopathology*; D013314:Stress, Mechanical