Response to: Pardossi-Piquard et al., "Presenilin-Dependent Transcriptional Control of the Abeta-Degrading Enzyme Neprilysin by Intracellular Domains of betaAPP and APLP." Neuron 46, 541-554.
Chen
Allen C
AC
Center for Neurologic Diseases, Brigham and Women's Hospital and Harvard Medial School, Boston, MA 02115, USA.
Selkoe
Dennis J
DJ
D016564:Amyloid beta-Protein Precursor / Q000378:metabolism*; D000818:Animals; D002478:Cells, Cultured; D004622:Embryo, Mammalian; D004791:Enzyme Inhibitors / Q000494:pharmacology; D005347:Fibroblasts; D006801:Humans; D051379:Mice; D018345:Mice, Knockout; D015260:Neprilysin / Q000378:metabolism*; D053763:Presenilins / Q000502:physiology*; D017434:Protein Structure, Tertiary; D014162:Transfection
C489697:APLP1 protein, human; D016564:Amyloid beta-Protein Precursor; D004791:Enzyme Inhibitors; D053763:Presenilins; D015260:Neprilysin