Abnormal expression of cardiac neural crest cells in heart development: a different hypothesis for the etiopathogenesis of Brugada syndrome.
Elizari
Marcelo Víctor
MV
Division of Cardiology, Ramos Mejía Hospital, Buenos Aires, Argentina. elizarimv@fibertel.com.ar
Levi
Raúl
R
Acunzo
Rafael Salvador
RS
Chiale
Pablo Ambrosio
PA
Civetta
María M
MM
Ferreiro
Marcela
M
Sicouri
Serge
S
D000200:Action Potentials; D000818:Animals; D053840:Brugada Syndrome / Q000196:embryology* / Q000209:etiology / Q000473:pathology* / Q000503:physiopathology; D002465:Cell Movement; D018031:Connexin 43 / Q000096:biosynthesis; D004562:Electrocardiography; D017629:Gap Junctions; D006330:Heart Defects, Congenital / Q000196:embryology / Q000209:etiology / Q000473:pathology / Q000503:physiopathology; D006801:Humans; D009432:Neural Crest / Q000002:abnormalities* / Q000378:metabolism / Q000503:physiopathology; D010641:Phenotype; D015222:Sodium Channels
D018031:Connexin 43; D015222:Sodium Channels