title: Mutant mice provide new insight into the role of (mis-)glycation in IgA nephropathy and other glomerular diseases.
authors: Department of Nephrology and Clinical Immunology, University Hospital Aachen, Pauwelsstrasse 30, D-52057 Aachen, Germany. juergen.floege@rwth-aachen.de
authors: Jürgen
authors: J
authors: Floege
authors: Frank
authors: F
authors: Eitner
authors: Jonathan
authors: J
authors: Barratt
authors: Alice C
authors: AC
authors: Smith
authors: John
authors: J
authors: Feehally
mesh_terms: D000818:Animals; D050260:Carbohydrate Metabolism / Q000235:genetics*; D004195:Disease Models, Animal; D005700:Galactosyltransferases / Q000235:genetics; D005922:Glomerulonephritis, IGA / Q000201:enzymology / Q000235:genetics* / Q000378:metabolism*; D006031:Glycosylation; D006801:Humans; D051379:Mice; D017354:Point Mutation
chemical_list: C515597:C1galt1 protein, mouse; D005700:Galactosyltransferases