title: Is angiotensin II a direct mediator of left ventricular hypertrophy? Time for another look.
authors: Laboratory of Molecular Biochemistry of Hypertension, Clinical Research Institute of Montreal, Montreal, Quebec, Canada. reudelt@ircm.qc.ca
authors: Timothy L
authors: TL
authors: Reudelhuber
authors: Kenneth E
authors: KE
authors: Bernstein
authors: Patrick
authors: P
authors: Delafontaine
mesh_terms: D000804:Angiotensin II / Q000502:physiology*; D000818:Animals; D001794:Blood Pressure / Q000502:physiology; D006801:Humans; D006973:Hypertension / Q000503:physiopathology; D017379:Hypertrophy, Left Ventricular / Q000209:etiology* / Q000235:genetics / Q000503:physiopathology*; D051379:Mice; D008822:Mice, Transgenic; D011401:Promoter Regions, Genetic / Q000235:genetics / Q000502:physiology; D044140:Receptor, Angiotensin, Type 1 / Q000502:physiology; D012084:Renin-Angiotensin System / Q000502:physiology; D014661:Vasoconstriction / Q000502:physiology; D024723:Ventricular Myosins / Q000235:genetics / Q000502:physiology
chemical_list: D044140:Receptor, Angiotensin, Type 1; D000804:Angiotensin II; D024723:Ventricular Myosins