title: NCX current in the murine embryonic heart: development-dependent regulation by Na+.
abstract: Due to its high functional expression, I(NCX) may serve as an important mechanism to ensure intracellular Ca2+ homeostasis, especially during the early embryonic stage. At the fetal stage I(NCX) density is significantly decreased but underlies regulatory processes, that is, regulation by Na+.
authors: Institute of Neurophysiology, University of Cologne, Robert-Koch-Str. 39, D-50931 Cologne, Germany. akp72@uni-koeln.de
authors: Michael
authors: M
authors: Reppel
authors: Hannes
authors: H
authors: Reuter
authors: Philipp
authors: P
authors: Sasse
authors: Jürgen
authors: J
authors: Hescheler
authors: Bernd K
authors: BK
authors: Fleischmann
mesh_terms: D000818:Animals; D002478:Cells, Cultured; D047108:Embryonic Development; D006321:Heart / Q000196:embryology* / Q000502:physiology*; D006706:Homeostasis; D051379:Mice; D012964:Sodium / Q000378:metabolism*; D019831:Sodium-Calcium Exchanger / Q000502:physiology*
chemical_list: D019831:Sodium-Calcium Exchanger; D012964:Sodium