The increase of apolipoprotein A-V during postprandial lipemia parallels the response of triglyceride-rich lipoproteins in type 2 diabetes: no relationship between apoA-V and postheparin plasma lipolytic activity.
Kahri
Juhani
J
Division of Cardiology, Helsinki University Hospital, Biomedicum, Helsinki, Finland.
Fruchart-Najib
Jamila
J
Matikainen
Niina
N
Fruchart
Jean-Charles
JC
Vakkilainen
Juha
J
Taskinen
Marja-Riitta
MR
D000818:Animals; D000072040:Apolipoprotein A-V; D001054:Apolipoproteins A / Q000097:blood* / Q000172:deficiency / Q000235:genetics; D003924:Diabetes Mellitus, Type 2 / Q000097:blood*; D006801:Humans; D006949:Hyperlipidemias / Q000097:blood*; D008066:Lipolysis / Q000502:physiology*; D008074:Lipoproteins / Q000097:blood*; D051379:Mice; D018345:Mice, Knockout; D008822:Mice, Transgenic; D019518:Postprandial Period; D014280:Triglycerides / Q000097:blood*
C437704:APOA5 protein, human; D000072040:Apolipoprotein A-V; D001054:Apolipoproteins A; D008074:Lipoproteins; D014280:Triglycerides