Arsenic: extension of its endocrine disruption potential to interference with estrogen receptor-mediated signaling.
Watson
Walter H
WH
Division of Toxicological Sciences, Department of Environmental Health Sciences, Johns Hopkins Bloomberg School of Public Health, Baltimore, Maryland 21205, USA. wwatson@jhsph.edu
Yager
James D
JD
D000818:Animals; D001151:Arsenic / Q000633:toxicity*; D052244:Endocrine Disruptors / Q000633:toxicity*; D006801:Humans; D011960:Receptors, Estrogen / Q000187:drug effects*; D015398:Signal Transduction / Q000187:drug effects*
D052244:Endocrine Disruptors; D011960:Receptors, Estrogen; D001151:Arsenic