On the viral hypothesis of multiple sclerosis: participation of varicella-zoster virus.
Recent studies, including our own, have accumulated evidence suggesting the etiological participation of varicella-zoster virus in multiple sclerosis. If confirmed, complex issues of individual susceptibility and immunopathogenesis would have to be unveiled.
Sotelo
Julio
J
Neuroimmunology Unit, National Institute of Neurology and Neurosurgery, Mexico City, Mexico. jsotelo@servidor.unam.mx
D002644:Chickenpox / Q000150:complications* / Q000276:immunology* / Q000503:physiopathology; D005724:Ganglia / Q000276:immunology / Q000821:virology; D020022:Genetic Predisposition to Disease / Q000235:genetics; D006562:Herpes Zoster / Q000276:immunology* / Q000503:physiopathology / Q000821:virology*; D014645:Herpesvirus 3, Human / Q000235:genetics / Q000276:immunology*; D006801:Humans; D009103:Multiple Sclerosis / Q000276:immunology* / Q000503:physiopathology / Q000821:virology*; D020529:Multiple Sclerosis, Relapsing-Remitting / Q000276:immunology / Q000503:physiopathology / Q000821:virology; D014612:Vaccines / Q000276:immunology / Q000627:therapeutic use; D019562:Viral Load
D014612:Vaccines