The enteroinsular axis may mediate the diabetogenic effects of TCF7L2 polymorphisms.
Nauck
M A
MA
Diabeteszentrum Bad Lauterberg, Kirchberg 21, D-37431, Bad Lauterberg, Harz, Germany. M.Nauck@diabeteszentrum.de
Meier
J J
JJ
D048909:Diabetes Complications / Q000235:genetics; D003920:Diabetes Mellitus / Q000235:genetics*; D005749:Gastric Inhibitory Polypeptide / Q000502:physiology; D020022:Genetic Predisposition to Disease; D052216:Glucagon-Like Peptide 1 / Q000502:physiology; D006801:Humans; D006943:Hyperglycemia / Q000209:etiology / Q000235:genetics; D007328:Insulin / Q000378:metabolism / Q000502:physiology*; D000078790:Insulin Secretion; D007413:Intestinal Mucosa / Q000378:metabolism; D007422:Intestines / Q000502:physiology*; D008954:Models, Biological; D020641:Polymorphism, Single Nucleotide / Q000502:physiology*; D015398:Signal Transduction / Q000502:physiology; D051820:TCF Transcription Factors / Q000235:genetics* / Q000378:metabolism; D058118:Transcription Factor 7-Like 2 Protein
D007328:Insulin; D051820:TCF Transcription Factors; C544894:TCF7L2 protein, human; D058118:Transcription Factor 7-Like 2 Protein; D005749:Gastric Inhibitory Polypeptide; D052216:Glucagon-Like Peptide 1