When the lung is stretched, could it be thrombospondin via TGFbeta1 peptide activation?
Warburton
David
D
Developmental Biology and Regenerative Medicine Program, Saban Reasearch Institute, Children's Hospital Los AAngeles, Keck School of Dentistry, University of Southern California, Los Angeles, CA 90027, USA. dwarbuton@chla.usc.edu
Kaartinen
Vesa
V
D000818:Animals; D000831:Animals, Newborn; D049109:Cell Proliferation; D051766:Early Growth Response Protein 1 / Q000378:metabolism; D004847:Epithelial Cells / Q000378:metabolism; D005347:Fibroblasts / Q000378:metabolism; D018507:Gene Expression Regulation, Developmental; D005865:Gestational Age; D006801:Humans; D008168:Lung / Q000196:embryology / Q000254:growth & development / Q000378:metabolism* / Q000473:pathology; D008171:Lung Diseases / Q000235:genetics / Q000378:metabolism; D040542:Mechanotransduction, Cellular / Q000235:genetics; D051379:Mice; D011661:Pulmonary Stretch Receptors / Q000378:metabolism*; D012333:RNA, Messenger / Q000378:metabolism; D012756:Sheep; D019700:Thrombospondin 1 / Q000235:genetics / Q000378:metabolism*; D013997:Time Factors; D014132:Trachea / Q000601:surgery; D014158:Transcription, Genetic; D053773:Transforming Growth Factor beta1 / Q000235:genetics / Q000378:metabolism*
D051766:Early Growth Response Protein 1; D012333:RNA, Messenger; D019700:Thrombospondin 1; D053773:Transforming Growth Factor beta1