An Il-lumen-ating look at ryanodine receptor modulation by disruption in triadin and calsequestrin interactions in cardiac myocytes.
Ramay
Hena R
HR
Department of Pharmacology and Systems Therapeutics, Mount Sinai School of Medicine, One Gustave Levy Place, Box 1215, New York, NY 10029, USA. hena.ramay@mssm.edu
Fabris
Frank
F
Noel
Onika
O
Sarkar
Amrita
A
D000818:Animals; D002118:Calcium / Q000378:metabolism; D002155:Calsequestrin / Q000378:metabolism*; D002352:Carrier Proteins / Q000378:metabolism*; D002478:Cells, Cultured; D008051:Lipid Bilayers / Q000378:metabolism; D009124:Muscle Proteins / Q000378:metabolism*; D032383:Myocytes, Cardiac / Q000166:cytology / Q000378:metabolism*; D051381:Rats; D019837:Ryanodine Receptor Calcium Release Channel / Q000378:metabolism*; D012519:Sarcoplasmic Reticulum / Q000378:metabolism*
D002155:Calsequestrin; D002352:Carrier Proteins; D008051:Lipid Bilayers; D009124:Muscle Proteins; D019837:Ryanodine Receptor Calcium Release Channel; C070048:triadin; D002118:Calcium