An Il-lumen-ating look at ryanodine receptor modulation by disruption in triadin and calsequestrin interactions in cardiac myocytes.
Department of Pharmacology and Systems Therapeutics, Mount Sinai School of Medicine, One Gustave Levy Place, Box 1215, New York, NY 10029, USA. hena.ramay@mssm.edu
Hena R
HR
Ramay
Frank
F
Fabris
Onika
O
Noel
Amrita
A
Sarkar
D000818:Animals; D002118:Calcium / Q000378:metabolism; D002155:Calsequestrin / Q000378:metabolism*; D002352:Carrier Proteins / Q000378:metabolism*; D002478:Cells, Cultured; D008051:Lipid Bilayers / Q000378:metabolism; D009124:Muscle Proteins / Q000378:metabolism*; D032383:Myocytes, Cardiac / Q000166:cytology / Q000378:metabolism*; D051381:Rats; D019837:Ryanodine Receptor Calcium Release Channel / Q000378:metabolism*; D012519:Sarcoplasmic Reticulum / Q000378:metabolism*
D002155:Calsequestrin; D002352:Carrier Proteins; D008051:Lipid Bilayers; D009124:Muscle Proteins; D019837:Ryanodine Receptor Calcium Release Channel; C070048:triadin; D002118:Calcium