title: Calcium channel modulation as an all-purpose mechanism for short-term synaptic plasticity.
abstract: The paper by Mochida et al. in this issue of Neuron proposes that synaptic facilitation, depression, and augmentation all arise from calcium-dependent regulation of calcium channels in the presynaptic terminal. Their proposal provides a unifying explanation for several forms of short-term presynaptic plasticity.
authors.affiliation: Department of Neurobiology, Duke University Medical Center, Box 3209, Durham, NC 27710, USA.
authors.forename: Qing
authors.initials: Q
authors.lastname: Cheng
authors.forename: George J
authors.initials: GJ
authors.lastname: Augustine
mesh_terms: D000818:Animals; D015220:Calcium Channels / Q000502:physiology*; D009473:Neuronal Plasticity / Q000502:physiology*; D013569:Synapses / Q000502:physiology*
chemical_list: D015220:Calcium Channels