The role of SLC26A6-mediated chloride/oxalate exchange in causing susceptibility to nephrolithiasis.
Soleimani
Manoocher
M
Department of Medicine, University of Cincinnati, Cincinnati Medical Center, 231 Albert Sabin Way, MSB G259, Cincinnati, OH 45267-0585, USA. manoocher.soleimani@uc.edu
D000818:Animals; D017920:Antiporters / Q000235:genetics* / Q000378:metabolism; D002712:Chlorides / Q000378:metabolism*; D020022:Genetic Predisposition to Disease / Q000235:genetics*; D006801:Humans; D026901:Membrane Transport Proteins / Q000235:genetics* / Q000378:metabolism; D051379:Mice; D053040:Nephrolithiasis / Q000235:genetics* / Q000378:metabolism*; D010070:Oxalates / Q000378:metabolism*; D000076988:Sulfate Transporters
D017920:Antiporters; D002712:Chlorides; D026901:Membrane Transport Proteins; D010070:Oxalates; C439529:SLC26A6 protein, human; C466900:Slc26a6 protein, mouse; D000076988:Sulfate Transporters