Spontaneous resolution of hepatitis C virus infection is not due to a mutation at Cys-508 of MAVS/VISA/IPS-1/CARDIF.
Huang
Kenneth H
KH
Bruneau
Julie
J
Shoukry
Naglaa
N
Bernard
Nicole F
NF
D048868:Adaptor Proteins, Signal Transducing / Q000737:chemistry / Q000235:genetics* / Q000378:metabolism; D003545:Cysteine / Q000235:genetics; D016174:Hepacivirus / Q000235:genetics / Q000302:isolation & purification / Q000472:pathogenicity*; D006526:Hepatitis C / Q000276:immunology* / Q000821:virology; D019698:Hepatitis C, Chronic / Q000821:virology; D006801:Humans; D007113:Immunity, Innate; D046912:Multiprotein Complexes / Q000737:chemistry / Q000235:genetics* / Q000378:metabolism; D009154:Mutation; D016133:Polymerase Chain Reaction; D017422:Sequence Analysis, DNA; D017361:Viral Nonstructural Proteins / Q000378:metabolism; D012697:Serine Endopeptidases; D043583:Nucleoside-Triphosphatase; D053487:DEAD-box RNA Helicases; D000085242:Viral Proteases
D048868:Adaptor Proteins, Signal Transducing; D003545:Cysteine; D046912:Multiprotein Complexes; D017361:Viral Nonstructural Proteins; C505371:MAVS protein, human; C084422:NS3 protein, hepatitis C virus; D012697:Serine Endopeptidases; D043583:Nucleoside-Triphosphatase; D053487:DEAD-box RNA Helicases; D000085242:Viral Proteases; C093542:NS4 protein, hepatitis C virus