The role of mitochondria in status epilepticus.
Cock
Hannah
H
Epilepsy Group, Department of Cardiac and Vascular Sciences, St. Georges, University of London and Atkinson Morley Regional Neuroscience Centre, St. Georges Hospital, London, United Kingdom. hcock@sgul.ac.uk
D000818:Animals; D020169:Caspases / Q000378:metabolism; D002415:Cats; D016923:Cell Death / Q000502:physiology; D004195:Disease Models, Animal; D005609:Free Radicals / Q000378:metabolism; D006624:Hippocampus / Q000187:drug effects / Q000378:metabolism / Q000503:physiopathology; D006801:Humans; D007608:Kainic Acid; D008928:Mitochondria / Q000378:metabolism / Q000502:physiology*; D028361:Mitochondrial Diseases / Q000378:metabolism / Q000503:physiopathology*; D009474:Neurons / Q000378:metabolism; D018384:Oxidative Stress / Q000502:physiology; D051381:Rats; D013226:Status Epilepticus / Q000139:chemically induced / Q000378:metabolism / Q000503:physiopathology*
D005609:Free Radicals; D020169:Caspases; D007608:Kainic Acid