title: [Homocysteine and rheumatic disease].
abstract: In the last decade hyperhomocysteinemia has become an element of great interest as a recognized factor of independent risk for atherotrombosis. Moreover a role has been suggested in some different diseases, like rheumatic ones. Hyperhomocysteinemia could represent a mechanism of amplification of vascular damage in rheumatic disease, therefore it could interact with treatements. Moreover it could contribute to explain the high incidence of cardiovascular events, than they do not find support in traditional risk factors.
authors: Dipartimento di Medicina Clinica e Sperimentale, Università, Verona.
authors: Ilaria
authors: I
authors: Tinazzi
authors: Paola
authors: P
authors: Caramaschi
authors: Giuseppe
authors: G
authors: Patuzzo
authors: Lisa Maria
authors: LM
authors: Bambara
authors: Domenico
authors: D
authors: Biasi
mesh_terms: D001172:Arthritis, Rheumatoid / Q000209:etiology; D001528:Behcet Syndrome / Q000209:etiology; D002318:Cardiovascular Diseases / Q000453:epidemiology / Q000209:etiology* / Q000503:physiopathology; D016022:Case-Control Studies; D004730:Endothelium, Vascular / Q000503:physiopathology; D005260:Female; D006710:Homocysteine / Q000097:blood / Q000378:metabolism; D006801:Humans; D020138:Hyperhomocysteinemia / Q000150:complications* / Q000453:epidemiology / Q000503:physiopathology; D007231:Infant, Newborn; D008297:Male; D015201:Meta-Analysis as Topic; D011446:Prospective Studies; D012216:Rheumatic Diseases / Q000453:epidemiology / Q000209:etiology* / Q000503:physiopathology; D012307:Risk Factors; D012595:Scleroderma, Systemic / Q000209:etiology
chemical_list: D006710:Homocysteine