title: Dioxin may promote inflammation-related development of endometriosis.
abstract: Laboratory and population-based studies suggest that exposure to environmental toxicants may be one of several triggers for the development of endometriosis. We discuss evidence that modulation of the endometrial endocrine-immune interface could mechanistically link toxicant exposure to the development of this disease.
authors.affiliation: Women's Reproductive Health Research Center, Department of Obstetrics and Gynecology, Vanderbilt University School of Medicine, Nashville, Tennessee 37232, USA.
authors.forename: Kaylon L
authors.initials: KL
authors.lastname: Bruner-Tran
authors.forename: Grant R
authors.initials: GR
authors.lastname: Yeaman
authors.forename: Marta A
authors.initials: MA
authors.lastname: Crispens
authors.forename: Toshio M
authors.initials: TM
authors.lastname: Igarashi
authors.forename: Kevin G
authors.initials: KG
authors.lastname: Osteen
mesh_terms: D000818:Animals; D004147:Dioxins / Q000633:toxicity*; D004195:Disease Models, Animal; D004715:Endometriosis / Q000139:chemically induced* / Q000473:pathology / Q000503:physiopathology; D004717:Endometrium / Q000502:physiology; D004781:Environmental Exposure / Q000009:adverse effects; D005260:Female; D006801:Humans; D007249:Inflammation / Q000139:chemically induced*; D008954:Models, Biological
chemical_list: D004147:Dioxins