Dioxin may promote inflammation-related development of endometriosis.
Laboratory and population-based studies suggest that exposure to environmental toxicants may be one of several triggers for the development of endometriosis. We discuss evidence that modulation of the endometrial endocrine-immune interface could mechanistically link toxicant exposure to the development of this disease.
Bruner-Tran
Kaylon L
KL
Women's Reproductive Health Research Center, Department of Obstetrics and Gynecology, Vanderbilt University School of Medicine, Nashville, Tennessee 37232, USA.
Yeaman
Grant R
GR
Crispens
Marta A
MA
Igarashi
Toshio M
TM
Osteen
Kevin G
KG
D000818:Animals; D004147:Dioxins / Q000633:toxicity*; D004195:Disease Models, Animal; D004715:Endometriosis / Q000139:chemically induced* / Q000473:pathology / Q000503:physiopathology; D004717:Endometrium / Q000502:physiology; D004781:Environmental Exposure / Q000009:adverse effects; D005260:Female; D006801:Humans; D007249:Inflammation / Q000139:chemically induced*; D008954:Models, Biological
D004147:Dioxins