[Mechanisms implicated in the progression of IgA nephropathy].
Kawamura
Tetsuya
T
D000818:Animals; D018932:Chemokine CCL2 / Q000235:genetics; D016207:Cytokines / Q000502:physiology; D018450:Disease Progression; D005920:Glomerular Mesangium / Q000276:immunology / Q000473:pathology; D005922:Glomerulonephritis, IGA / Q000209:etiology*; D006801:Humans; D006973:Hypertension / Q000150:complications; D007070:Immunoglobulin A / Q000378:metabolism; D018836:Inflammation Mediators / Q000502:physiology; D007333:Insulin Resistance; D007684:Kidney Tubules / Q000473:pathology; D008264:Macrophages / Q000276:immunology; D007703:Peptidyl-Dipeptidase A / Q000235:genetics; D050199:Podocytes; D011110:Polymorphism, Genetic; D011507:Proteinuria / Q000150:complications; D019713:Receptors, CCR5 / Q000235:genetics; D012307:Risk Factors
C111835:CCL2 protein, human; D018932:Chemokine CCL2; D016207:Cytokines; D007070:Immunoglobulin A; D018836:Inflammation Mediators; D019713:Receptors, CCR5; D007703:Peptidyl-Dipeptidase A