[Role of jak/STAT3 and PI3-K/Akt pathways in liver injury and regeneration].
Ozaki
Michitaka
M
Department of Molecular Surgery, Hokkaido University School of Medicine, Sapporo, Japan.
D000818:Animals; D006801:Humans; D008099:Liver / Q000473:pathology* / Q000502:physiology*; D008115:Liver Regeneration / Q000235:genetics* / Q000502:physiology*; D051379:Mice; D019869:Phosphatidylinositol 3-Kinases / Q000502:physiology*; D017346:Protein Serine-Threonine Kinases / Q000502:physiology; D000081382:Pyruvate Dehydrogenase Acetyl-Transferring Kinase; D050796:STAT3 Transcription Factor / Q000502:physiology*; D015398:Signal Transduction / Q000502:physiology*
D019869:Phosphatidylinositol 3-Kinases; D017346:Protein Serine-Threonine Kinases; D000081382:Pyruvate Dehydrogenase Acetyl-Transferring Kinase; D050796:STAT3 Transcription Factor; C494087:Stat3 protein, mouse