Neuroinflammation as a neurotoxic mechanism in alcoholism: commentary on "Increased MCP-1 and microglia in various regions of human alcoholic brain".
Sullivan
Edith V
EV
Department of Psychiatry and Behavioral Sciences, Stanford University School of Medicine, 401 Quarry Road, Stanford, CA 94305, USA. edie@stanford.edu
Zahr
Natalie M
NM
D020268:Alcohol-Induced Disorders, Nervous System / Q000276:immunology / Q000473:pathology / Q000503:physiopathology*; D001921:Brain / Q000187:drug effects* / Q000473:pathology / Q000503:physiopathology; D002492:Central Nervous System Depressants / Q000633:toxicity; D018932:Chemokine CCL2 / Q000187:drug effects* / Q000276:immunology; D004660:Encephalitis / Q000139:chemically induced* / Q000276:immunology / Q000503:physiopathology; D000431:Ethanol / Q000633:toxicity; D005911:Gliosis / Q000139:chemically induced / Q000276:immunology / Q000503:physiopathology; D017628:Microglia / Q000187:drug effects* / Q000276:immunology; D009410:Nerve Degeneration / Q000139:chemically induced / Q000276:immunology / Q000503:physiopathology
C111835:CCL2 protein, human; D002492:Central Nervous System Depressants; D018932:Chemokine CCL2; D000431:Ethanol