The best medicine: exercise training normalizes chemosensitivity and sympathoexcitation in heart failure.
Stickland
Michael K
MK
Dept. of Medicine, Health Sciences Centre, Univ. of Alberta, Edmonton, Alberta, Canada T6G 2B7. michael.stickland@ualberta.ca
Miller
Jordan D
JD
D000200:Action Potentials; D000804:Angiotensin II / Q000378:metabolism; D000818:Animals; D002344:Carotid Body / Q000201:enzymology / Q000503:physiopathology*; D002908:Chronic Disease; D005081:Exercise Therapy; D006333:Heart Failure / Q000209:etiology / Q000378:metabolism / Q000503:physiopathology / Q000628:therapy*; D006439:Hemodynamics; D006801:Humans; D007668:Kidney / Q000294:innervation*; D009569:Nitric Oxide / Q000378:metabolism; D052248:Nitric Oxide Synthase Type I / Q000037:antagonists & inhibitors / Q000378:metabolism; D005082:Physical Exertion; D044140:Receptor, Angiotensin, Type 1 / Q000378:metabolism; D012018:Reflex; D015398:Signal Transduction; D013564:Sympathetic Nervous System / Q000503:physiopathology*
D044140:Receptor, Angiotensin, Type 1; D000804:Angiotensin II; D009569:Nitric Oxide; D052248:Nitric Oxide Synthase Type I