title: [Hemostasis and maintenange of the organism vitamin C].
abstract: The analysis of the literature on communication of a hemostasis and vitamin C has allowed to assert, that its deficiency accelerates lipid peroxidation and reduces potential at porpoises, and it conducts to activation platelets, to acceleration of continuous intravascular blood coagulation of and to reduction in tolerance to thrombin. High dozes of vitamin C influences a hemostasis strengthens lipid peroxidation similarly to prooxidizers. Entering high dozes of vitamin C at the statuses menacing weith thromboses, it is desirable to supervise a level of markers of intravascular blood coagulation in plasma.
authors.forename: A Sh
authors.initials: ASh
authors.lastname: Byshevskiĭ
authors.forename: S I
authors.initials: SI
authors.lastname: Mataev
authors.forename: A Iu
authors.initials: AIu
authors.lastname: Rudzevich
authors.forename: E M
authors.initials: EM
authors.lastname: Shpovalova
mesh_terms: D000818:Animals; D001205:Ascorbic Acid / Q000008:administration & dosage / Q000009:adverse effects / Q000378:metabolism*; D001206:Ascorbic Acid Deficiency / Q000378:metabolism; D001777:Blood Coagulation / Q000187:drug effects / Q000502:physiology*; D001792:Blood Platelets / Q000378:metabolism*; D004211:Disseminated Intravascular Coagulation / Q000139:chemically induced / Q000378:metabolism; D006706:Homeostasis; D006801:Humans; D015227:Lipid Peroxidation / Q000187:drug effects / Q000502:physiology*; D015539:Platelet Activation / Q000187:drug effects / Q000502:physiology*; D013917:Thrombin / Q000378:metabolism; D013927:Thrombosis / Q000139:chemically induced / Q000378:metabolism
chemical_list: D013917:Thrombin; D001205:Ascorbic Acid