Exercise pretraining attenuates endotoxin-induced hemodynamic alteration in type I diabetic rats.
D000818:Animals; D001786:Blood Glucose / Q000378:metabolism; D015153:Blotting, Western; D003921:Diabetes Mellitus, Experimental / Q000097:blood / Q000503:physiopathology*; D003922:Diabetes Mellitus, Type 1 / Q000097:blood / Q000503:physiopathology*; D004731:Endotoxins / Q000633:toxicity*; D050884:HSP72 Heat-Shock Proteins / Q000378:metabolism; D006439:Hemodynamics / Q000187:drug effects*; D008070:Lipopolysaccharides / Q000633:toxicity; D008297:Male; D008526:Medulla Oblongata / Q000378:metabolism; D009206:Myocardium / Q000378:metabolism; D019001:Nitric Oxide Synthase / Q000096:biosynthesis; D010805:Physical Conditioning, Animal / Q000502:physiology*; D051381:Rats; D017208:Rats, Wistar; D017552:Solitary Nucleus / Q000378:metabolism; D014409:Tumor Necrosis Factor-alpha / Q000097:blood
D001786:Blood Glucose; D004731:Endotoxins; D050884:HSP72 Heat-Shock Proteins; D008070:Lipopolysaccharides; D014409:Tumor Necrosis Factor-alpha; D019001:Nitric Oxide Synthase