Glucose-induced insulin secretion: is the small G-protein Rab27A the mediator of the K(ATP) channel-independent effect?
Ullrich
Susanne
S
University of Tübingen, Department of Internal Medicine, Division of Endo-crinology, Diabetology, Angiology, Nephrology and Clinical Chemistry, Tübingen, Germany. susanne.ullrich@med.unituebingen.de
D000818:Animals; D005089:Exocytosis; D005947:Glucose / Q000494:pharmacology*; D007328:Insulin / Q000378:metabolism*; D000078790:Insulin Secretion; D050417:Insulin-Secreting Cells / Q000187:drug effects / Q000378:metabolism; D054086:KATP Channels / Q000378:metabolism*; D008564:Membrane Potentials; D051379:Mice; D020691:rab GTP-Binding Proteins / Q000172:deficiency / Q000235:genetics / Q000378:metabolism*; D000075745:rab27 GTP-Binding Proteins
D007328:Insulin; D054086:KATP Channels; D000075745:rab27 GTP-Binding Proteins; C512962:Rab27a protein, mouse; D020691:rab GTP-Binding Proteins; D005947:Glucose