title: The effect of aspirin on Gomori-positive glia in mouse brain.
abstract: Aspirin (acetylsalicylic acid, ASA) treatment resulted in a significant decrease in the amount of the sulfur-rich Gomori-positive material present in the cytoplasm of periventricular glia. It also caused the accumulation of the Gomori-positive neurosecretory material in the supraoptic and paraventricular nuclei and, most pronounced, in the neurosecretory axons of the paraventricular- and supraoptic-neurophypophysial tract.
authors.affiliation: Departament of Human Developmental Biology, Collegium Medicum, Jagiellonian University, Kopernika 7, 31-034 Kraków, Poland. mbsura@cyf-kr.edu.pl
authors.forename: Piotr
authors.initials: P
authors.lastname: Sura
authors.forename: Zbigniew
authors.initials: Z
authors.lastname: Srebro
authors.forename: Bogdan
authors.initials: B
authors.lastname: Wiliński
authors.forename: Marta
authors.initials: M
authors.lastname: Góralska
mesh_terms: D000818:Animals; D001241:Aspirin / Q000494:pharmacology*; D001921:Brain / Q000166:cytology*; D003593:Cytoplasm / Q000378:metabolism; D006416:Hematoxylin; D051379:Mice; D009457:Neuroglia / Q000187:drug effects*; D013194:Staining and Labeling
chemical_list: D001241:Aspirin; D006416:Hematoxylin