The effect of aspirin on Gomori-positive glia in mouse brain.
Aspirin (acetylsalicylic acid, ASA) treatment resulted in a significant decrease in the amount of the sulfur-rich Gomori-positive material present in the cytoplasm of periventricular glia. It also caused the accumulation of the Gomori-positive neurosecretory material in the supraoptic and paraventricular nuclei and, most pronounced, in the neurosecretory axons of the paraventricular- and supraoptic-neurophypophysial tract.
Sura
Piotr
P
Departament of Human Developmental Biology, Collegium Medicum, Jagiellonian University, Kopernika 7, 31-034 Kraków, Poland. mbsura@cyf-kr.edu.pl
Srebro
Zbigniew
Z
Wiliński
Bogdan
B
Góralska
Marta
M
D000818:Animals; D001241:Aspirin / Q000494:pharmacology*; D001921:Brain / Q000166:cytology*; D003593:Cytoplasm / Q000378:metabolism; D006416:Hematoxylin; D051379:Mice; D009457:Neuroglia / Q000187:drug effects*; D013194:Staining and Labeling
D001241:Aspirin; D006416:Hematoxylin