Cerebral infarction and right ventricular noncompaction.
A 26-year-old man admitted with dysphasia and bilateral headache for three days. He had no known risk factors for stroke. Transthoracic two-dimensional echocardiography showed prominent trabeculations, with deep intertrabecular recesses in apex of the right ventricle. The multi-detector-row spiral cerebral computerized tomography revealed a little infarction in bilateral temporal and frontal lobe. The main mechanisms of neurovascular complications in NVM may be related to development of thrombi in the intertrabecular space. Extensively trabeculated ventricle, depressed systolic function, and/or the development of atrial fibrillation are the causes for thrombus formation7. Embolic stroke has been only rarely described in association with NVM9. However, whether NVM is a risk factor for stroke is controversially discussed. Prevention of embolic complications is an important management issue, and several authors have recommended long-term prophylactic anticoagulation for patients in NVM with atrial fibrillation and severe systolic dysfunction. The field of neurovascular complications, and specially cerebral infarction, in NVM of right ventricular remains not yet fully understood.
Song
Ze-Zhou
ZZ