[Novel mechanism of herpes simplex virus type-I infection].
Satoh
Takeshi
T
Department of Immunochemistry, Research Institute for Microbial Diseases, Osaka University, 3-1, Yamadaoka, Suita, Osaka 565-0871, Japan.
Arase
Hisashi
H
D000818:Animals; D015815:Cell Adhesion Molecules / Q000378:metabolism; D006561:Herpes Simplex / Q000821:virology*; D018259:Herpesvirus 1, Human / Q000235:genetics / Q000472:pathogenicity*; D006801:Humans; D008562:Membrane Glycoproteins / Q000378:metabolism*; D000075983:Nectins; D011485:Protein Binding; D011971:Receptors, Immunologic / Q000378:metabolism*; D053240:Receptors, Tumor Necrosis Factor, Member 14 / Q000378:metabolism; D014759:Viral Envelope Proteins / Q000378:metabolism*
D015815:Cell Adhesion Molecules; D008562:Membrane Glycoproteins; D000075983:Nectins; C412263:PILRA protein, human; D011971:Receptors, Immunologic; D053240:Receptors, Tumor Necrosis Factor, Member 14; C506028:TNFRSF14 protein, human; D014759:Viral Envelope Proteins; C040495:glycoprotein B, Simplexvirus; C039447:glycoprotein D, Human herpesvirus 1