Diacylglycerol kinase alpha, from negative modulation of T cell activation to control of cancer progression.
Mérida
Isabel
I
Department of Immunology and Oncology, Centro Nacional de Biotecnología/CSIC, Darwin 3, Campus de Cantoblanco, E-28049 Madrid, Spain. imerida@cnb.csic.es
Avila-Flores
Antonia
A
García
Job
J
Merino
Ernesto
E
Almena
María
M
Torres-Ayuso
Pedro
P
D000818:Animals; D002465:Cell Movement; D049109:Cell Proliferation; D019852:Diacylglycerol Kinase / Q000235:genetics / Q000378:metabolism*; D004075:Diglycerides / Q000378:metabolism; D015971:Gene Expression Regulation, Enzymologic; D007527:Isoenzymes / Q000235:genetics / Q000378:metabolism; D008213:Lymphocyte Activation; D009369:Neoplasms / Q000473:pathology / Q000503:physiopathology; D010766:Phosphorylation; D011505:Protein-Tyrosine Kinases / Q000378:metabolism; D012333:RNA, Messenger / Q000235:genetics / Q000378:metabolism; D015398:Signal Transduction / Q000502:physiology; D013601:T-Lymphocytes / Q000502:physiology*; D016159:Tumor Suppressor Protein p53 / Q000378:metabolism
D004075:Diglycerides; D007527:Isoenzymes; D012333:RNA, Messenger; D016159:Tumor Suppressor Protein p53; D019852:Diacylglycerol Kinase; D011505:Protein-Tyrosine Kinases