Recruitment of 9-1-1 to sites of DNA damage through cell cycle-dependent processing of DNA lesions.
Lakin
Nicholas D
ND
Department of Biochemistry, University of Oxford, Oxford, UK. nicholas.lakin@ bioch.ox.ac.uk
D064007:Ataxia Telangiectasia Mutated Proteins; D002453:Cell Cycle; D018797:Cell Cycle Proteins / Q000378:metabolism*; D004249:DNA Damage; D004260:DNA Repair; D004277:DNA, Single-Stranded / Q000378:metabolism; D004268:DNA-Binding Proteins / Q000378:metabolism; D005092:Exonucleases / Q000378:metabolism*; D006801:Humans; D017346:Protein Serine-Threonine Kinases / Q000378:metabolism; D025521:Tumor Suppressor Proteins / Q000378:metabolism
D018797:Cell Cycle Proteins; D004277:DNA, Single-Stranded; D004268:DNA-Binding Proteins; C462949:HUS1B protein, human; D025521:Tumor Suppressor Proteins; C069930:rad9 protein; C576324:ATM protein, human; C576597:ATR protein, human; D064007:Ataxia Telangiectasia Mutated Proteins; D017346:Protein Serine-Threonine Kinases; D005092:Exonucleases; C113574:Rad1 protein, human