Regression of cerebral calcifications after endovascular treatment in a case of vein of Galen arteriovenous malformation.
The authors present the case of an infant harboring a vein of Galen arteriovenous malformation with conspicuous cerebral calcifications that progressively regressed after staged endovascular obliteration of the lesion. The role of venous hypertension and hydrocephalus secondary to the arteriovenous shunt are discussed to explain the formation and regression of the cerebral calcifications.
Bansal
Anshuman
A
Division of Interventional Neuroradiology, The John Hopkins Hospital, Baltimore, Maryland, USA.
Gailloud
Philippe
P
Jordan
Lori
L
Ruíz
Diego San Millán
DS
D002114:Calcinosis / Q000000981:diagnostic imaging / Q000503:physiopathology*; D002533:Cerebral Angiography; D002550:Cerebral Veins / Q000002:abnormalities / Q000601:surgery*; D004621:Embolization, Therapeutic / Q000379:methods*; D006801:Humans; D006849:Hydrocephalus / Q000209:etiology / Q000503:physiopathology; D006973:Hypertension / Q000209:etiology / Q000503:physiopathology; D007223:Infant; D008297:Male; D014057:Tomography, X-Ray Computed; D016896:Treatment Outcome; D054080:Vein of Galen Malformations / Q000000981:diagnostic imaging* / Q000503:physiopathology / Q000601:surgery*