Alternative splicing of TRIP8b diversifies its actions as an accessory subunit of neuronal HCN channels.
Braun
Andrew P
AP
Department of Physiology and Pharmacology, University of Calgary, Calgary, AB, CA. Email: abraun@ucalgary.ca
D017398:Alternative Splicing; D000818:Animals; D001923:Brain Chemistry; D054815:Cyclic Nucleotide-Gated Cation Channels; D006321:Heart / Q000502:physiology; D006801:Humans; D064428:Hyperpolarization-Activated Cyclic Nucleotide-Gated Channels; D008565:Membrane Proteins / Q000235:genetics* / Q000502:physiology; D051379:Mice; D009474:Neurons / Q000737:chemistry*; D000074401:Peroxins; D015221:Potassium Channels; D051381:Rats
D054815:Cyclic Nucleotide-Gated Cation Channels; D064428:Hyperpolarization-Activated Cyclic Nucleotide-Gated Channels; D008565:Membrane Proteins; D000074401:Peroxins; C501732:Pex5l protein, mouse; C523657:Pex5l protein, rat; D015221:Potassium Channels