Increased inotropism following PARP inhibition in the setting of myocardial reperfusion injury: another piece in the jigsaw?
Rehberg
Sebastian
S
Investigational Intensive Care Unit, Department of Anesthesiology, The University of Texas Medical Branch, Galveston, Texas, USA.
Thiemermann
Christoph
C
Traber
Daniel L
DL
D000818:Animals; D002316:Cardiotonic Agents / Q000627:therapeutic use*; D007211:Indoles / Q000494:pharmacology / Q000627:therapeutic use*; D009203:Myocardial Infarction / Q000188:drug therapy / Q000473:pathology; D015428:Myocardial Reperfusion Injury / Q000188:drug therapy* / Q000378:metabolism; D000067856:Poly(ADP-ribose) Polymerase Inhibitors; D011065:Poly(ADP-ribose) Polymerases / Q000378:metabolism; D013318:Stroke Volume / Q000187:drug effects; D013552:Swine
D002316:Cardiotonic Agents; C491685:INO 1001; D007211:Indoles; D000067856:Poly(ADP-ribose) Polymerase Inhibitors; D011065:Poly(ADP-ribose) Polymerases