[Novel spatiotemporal regulation of kinase signaling by ubiquitination: MAP kinase is activated through translocation of the signaling complex from receptor to cytosol].
Matsuzawa
Atsushi
A
Laboratory of Cell Signaling, Graduate School of Pharmaceutical Sciences, The University of Tokyo, Japan.
D000818:Animals; D053265:B-Cell Activation Factor Receptor / Q000502:physiology; D003600:Cytosol / Q000378:metabolism*; D051034:Inhibitor of Apoptosis Proteins / Q000502:physiology; D048728:MAP Kinase Kinase Kinase 1 / Q000378:metabolism / Q000502:physiology*; D046912:Multiprotein Complexes / Q000378:metabolism; D010766:Phosphorylation; D017346:Protein Serine-Threonine Kinases / Q000378:metabolism; D021381:Protein Transport; D018124:Receptors, Tumor Necrosis Factor / Q000502:physiology*; D015398:Signal Transduction / Q000502:physiology*; D047992:TNF Receptor-Associated Factor 2 / Q000502:physiology; D048008:TNF Receptor-Associated Factor 3 / Q000502:physiology; D054875:Ubiquitination; D000097782:NF-kappaB-Inducing Kinase
D053265:B-Cell Activation Factor Receptor; D051034:Inhibitor of Apoptosis Proteins; D048728:MAP Kinase Kinase Kinase 1; D046912:Multiprotein Complexes; D017346:Protein Serine-Threonine Kinases; D018124:Receptors, Tumor Necrosis Factor; D047992:TNF Receptor-Associated Factor 2; D048008:TNF Receptor-Associated Factor 3; D000097782:NF-kappaB-Inducing Kinase