title: How to shut down Ca2+-induced Ca2+ release?
authors.affiliation: Department of Physiology, University of Bern, Buehlplatz 5, 3012 Bern, Switzerland. niggli@pyl.unibe.ch
authors.forename: Ernst
authors.initials: E
authors.lastname: Niggli
mesh_terms: D000818:Animals; D001145:Arrhythmias, Cardiac / Q000378:metabolism; D002110:Caffeine / Q000494:pharmacology; D002118:Calcium / Q000378:metabolism; D020013:Calcium Signaling / Q000187:drug effects*; D002154:Calpain / Q000378:metabolism; D002155:Calsequestrin / Q000172:deficiency; D000697:Central Nervous System Stimulants / Q000494:pharmacology; D004285:Dogs; D006801:Humans; D008297:Male; D051379:Mice; D032383:Myocytes, Cardiac / Q000187:drug effects / Q000378:metabolism*; D019837:Ryanodine Receptor Calcium Release Channel / Q000378:metabolism*; D012519:Sarcoplasmic Reticulum / Q000187:drug effects / Q000378:metabolism*
chemical_list: D002155:Calsequestrin; D000697:Central Nervous System Stimulants; D019837:Ryanodine Receptor Calcium Release Channel; D002110:Caffeine; D002154:Calpain; D002118:Calcium