[Molecular mechanisms and physiological roles of DNA degradation].
Kawane
Kohki
K
Department of Medical Chemistry, Graduate School of Medicine, Kyoto University, Sakyo, Kyoto, Japan.
D000818:Animals; D017209:Apoptosis / Q000235:genetics / Q000502:physiology; D051017:Apoptosis Regulatory Proteins / Q000502:physiology*; D001172:Arthritis, Rheumatoid / Q000209:etiology; D002454:Cell Differentiation; D004247:DNA / Q000378:metabolism*; D003851:Deoxyribonucleases / Q000502:physiology*; D004706:Endodeoxyribonucleases / Q000172:deficiency / Q000502:physiology; D004912:Erythrocytes / Q000166:cytology; D019143:Evolution, Molecular; D053200:Fas-Associated Death Domain Protein / Q000502:physiology; D006801:Humans; D007908:Lens, Crystalline / Q000166:cytology / Q000378:metabolism; D008264:Macrophages / Q000201:enzymology / Q000378:metabolism; D051379:Mice; D015398:Signal Transduction / Q000502:physiology
D051017:Apoptosis Regulatory Proteins; D053200:Fas-Associated Death Domain Protein; C109664:caspase-activated DNase inhibitor; D004247:DNA; D003851:Deoxyribonucleases; D004706:Endodeoxyribonucleases; C109663:caspase-activated deoxyribonuclease; C019329:deoxyribonuclease II