Molecular basis of self-sustaining seizures and pharmacoresistance during status epilepticus: The receptor trafficking hypothesis revisited.
Wasterlain
Claude G
CG
Epilepsy Research Laboratory, VA Greater Los Angeles Health Care System, California 90073, USA. wasterla@ucla.edu
Liu
Hantao
H
Naylor
David E
DE
Thompson
Kerry W
KW
Suchomelova
Lucy
L
Niquet
Jerome
J
Mazarati
Audrey M
AM
Baldwin
Roger A
RA
D000818:Animals; D000927:Anticonvulsants / Q000494:pharmacology / Q000627:therapeutic use*; D004195:Disease Models, Animal; D004351:Drug Resistance; D004827:Epilepsy / Q000139:chemically induced / Q000503:physiopathology; D006624:Hippocampus / Q000503:physiopathology; D006801:Humans; D007150:Immunohistochemistry; D008094:Lithium; D008959:Models, Neurological; D010862:Pilocarpine; D051381:Rats; D011963:Receptors, GABA-A / Q000502:physiology*; D016194:Receptors, N-Methyl-D-Aspartate / Q000502:physiology; D013226:Status Epilepticus / Q000139:chemically induced / Q000188:drug therapy* / Q000503:physiopathology; D013569:Synapses / Q000502:physiology
D000927:Anticonvulsants; C546273:GABRB3 protein, human; C546279:GABRG2 protein, human; D011963:Receptors, GABA-A; D016194:Receptors, N-Methyl-D-Aspartate; D010862:Pilocarpine; D008094:Lithium