title: Cardiac I(to), KCNE2, and Brugada syndrome: promiscuous subunit interactions, or what happens in HEK cells stays in HEK cells?
authors.forename: Gordon F
authors.initials: GF
authors.lastname: Tomaselli
mesh_terms: D001145:Arrhythmias, Cardiac / Q000235:genetics* / Q000401:mortality; D053840:Brugada Syndrome / Q000235:genetics*; D002460:Cell Line; D002478:Cells, Cultured; D003001:Cloning, Molecular; D016757:Death, Sudden, Cardiac; D004594:Electrophysiology; D005822:Genetic Vectors; D006329:Heart Conduction System / Q000502:physiology; D006801:Humans; D007700:Kinetics; D051605:Kv Channel-Interacting Proteins / Q000235:genetics* / Q000502:physiology; D008564:Membrane Potentials / Q000502:physiology; D009154:Mutation; D018408:Patch-Clamp Techniques; D010641:Phenotype; D024642:Potassium Channels, Voltage-Gated / Q000235:genetics*; D051672:Shal Potassium Channels / Q000502:physiology; D014162:Transfection
chemical_list: C489619:KCNE2 protein, human; C496752:KCNIP2 protein, human; D051605:Kv Channel-Interacting Proteins; D024642:Potassium Channels, Voltage-Gated; D051672:Shal Potassium Channels