Dendritic tiling through TOR signalling.
Hong
Weizhe
W
Howard Hughes Medical Institute and Department of Biology, Stanford University, Stanford, CA, USA.
Luo
Liqun
L
D000818:Animals; D030801:Animals, Genetically Modified; D003712:Dendrites / Q000378:metabolism*; D004330:Drosophila; D029721:Drosophila Proteins / Q000378:metabolism / Q000502:physiology*; D006801:Humans; D008024:Ligands; D008954:Models, Biological; D009474:Neurons / Q000378:metabolism; D019869:Phosphatidylinositol 3-Kinases / Q000378:metabolism / Q000502:physiology*; D011494:Protein Kinases; D015398:Signal Transduction; D058570:TOR Serine-Threonine Kinases; D014157:Transcription Factors / Q000378:metabolism
D029721:Drosophila Proteins; D008024:Ligands; D019869:Phosphatidylinositol 3-Kinases; D011494:Protein Kinases; D058570:TOR Serine-Threonine Kinases; D014157:Transcription Factors; C493245:CRTC2 protein, human; C416303:target of rapamycin protein, Drosophila