Further characterization of proteolytic Clostridium botulinum type A5 reveals that neurotoxin formation is unaffected by loss of the cntR (botR) promoter sigma factor binding site.
Carter
Andrew T
AT
Mason
David R
DR
Grant
Kathie A
KA
Franciosa
Giovanna
G
Aureli
Paulo
P
Peck
Michael W
MW
D001426:Bacterial Proteins / Q000235:genetics*; D001665:Binding Sites; D003014:Clostridium botulinum / Q000378:metabolism*; D015169:Colony Count, Microbial; D004269:DNA, Bacterial / Q000737:chemistry / Q000235:genetics; D023061:Gene Order; D005798:Genes, Bacterial; D050296:Microbial Viability; D008969:Molecular Sequence Data; D009498:Neurotoxins / Q000096:biosynthesis*; D011401:Promoter Regions, Genetic; D017422:Sequence Analysis, DNA; D017384:Sequence Deletion; D012808:Sigma Factor / Q000378:metabolism*; D015534:Trans-Activators / Q000235:genetics*
D001426:Bacterial Proteins; C116103:BotR protein, Clostridium botulinum; D004269:DNA, Bacterial; D009498:Neurotoxins; D012808:Sigma Factor; D015534:Trans-Activators