The acute phase response inhibits reverse cholesterol transport.
Feingold
Kenneth R
KR
Metabolism Section, Department of Veterans Affairs Medical Center, University of California San Francisco, San Francisco, CA, USA. kenneth.feingold@ucsf.edu
Grunfeld
Carl
C
D000210:Acute-Phase Reaction / Q000097:blood / Q000139:chemically induced / Q000378:metabolism / Q000503:physiopathology*; D000818:Animals; D050197:Atherosclerosis / Q000503:physiopathology; D001647:Bile Acids and Salts / Q000378:metabolism; D001692:Biological Transport; D002784:Cholesterol / Q000097:blood / Q000378:metabolism*; D005243:Feces / Q000737:chemistry; D054501:Group II Phospholipases A2 / Q000097:blood; D006801:Humans; D008070:Lipopolysaccharides / Q000633:toxicity; D008074:Lipoproteins / Q000097:blood / Q000378:metabolism*; D008075:Lipoproteins, HDL / Q000097:blood / Q000502:physiology; D008099:Liver / Q000378:metabolism; D008264:Macrophages / Q000378:metabolism; D051379:Mice; D009195:Peroxidase / Q000097:blood; D000685:Serum Amyloid A Protein
D001647:Bile Acids and Salts; D008070:Lipopolysaccharides; D008074:Lipoproteins; D008075:Lipoproteins, HDL; D000685:Serum Amyloid A Protein; D002784:Cholesterol; D009195:Peroxidase; D054501:Group II Phospholipases A2