Sepsis-induced myocardial dysfunction: linking physiology and genomics.
Cohen
Rubin I
RI
D000818:Animals; D024722:Cardiac Myosins / Q000235:genetics*; D004734:Energy Metabolism / Q000235:genetics* / Q000502:physiology*; D015870:Gene Expression / Q000235:genetics*; D005786:Gene Expression Regulation / Q000235:genetics / Q000502:physiology; D006333:Heart Failure / Q000235:genetics* / Q000503:physiopathology*; D051379:Mice; D008815:Mice, Inbred Strains; D009200:Myocardial Contraction / Q000235:genetics* / Q000502:physiology*; D018995:Myosin Heavy Chains / Q000235:genetics*; D052247:Nitric Oxide Synthase Type II / Q000235:genetics*; D000071248:Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha; D018805:Sepsis / Q000235:genetics* / Q000503:physiopathology*; D015534:Trans-Activators / Q000235:genetics*; D014157:Transcription Factors; D014158:Transcription, Genetic / Q000235:genetics*; D020257:Ventricular Remodeling / Q000235:genetics / Q000502:physiology
D000071248:Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha; C485290:Ppargc1a protein, mouse; D015534:Trans-Activators; D014157:Transcription Factors; D052247:Nitric Oxide Synthase Type II; C496317:Nos2 protein, mouse; D024722:Cardiac Myosins; D018995:Myosin Heavy Chains