Malignant malaria and microangiopathies: merging mechanisms.
In this issue of Blood, Bridges and colleagues report that Plasmodium falciparum-infected erythrocytes are able to attach to the endothelial surface by binding platelet-decorated VWF strands in a CD36-dependent fashion, revealing a new mechanism for erythrocyte cytoadherence in malaria.(1).
López
José A
JA
Puget Sound Blood Center.