title: NR1 knockdown mice as a representative model of the glutamate hypothesis of schizophrenia.
authors.affiliation: University of Toronto, Department of Pharmacology and Toxicology, Toronto, ON, Canada. a.ramsey@utoronto.ca
authors.forename: Amy J
authors.initials: AJ
authors.lastname: Ramsey
mesh_terms: D000818:Animals; D001923:Brain Chemistry / Q000235:genetics*; D015536:Down-Regulation / Q000235:genetics; D018698:Glutamic Acid / Q000378:metabolism*; D051379:Mice; D018345:Mice, Knockout / Q000235:genetics*; D009154:Mutation / Q000235:genetics; D016194:Receptors, N-Methyl-D-Aspartate / Q000235:genetics*; D012559:Schizophrenia / Q000235:genetics*; D009435:Synaptic Transmission / Q000235:genetics
chemical_list: C409466:NR1 NMDA receptor; D016194:Receptors, N-Methyl-D-Aspartate; D018698:Glutamic Acid