Progressive renal injury from transgenic expression of human carbonic anhydrase IV folding mutants is enhanced by deficiency of p58IPK.
Datta
Rupak
R
Edward A Doisy Department of Biochemistry and Molecular Biology, Saint Louis University School of Medicine, St Louis, MO 63104, USA.
Shah
Gul N
GN
Rubbelke
Timothy S
TS
Waheed
Abdul
A
Rauchman
Michael
M
Goodman
Alan G
AG
Katze
Michael G
MG
Sly
William S
WS
D000818:Animals; D017209:Apoptosis; D001483:Base Sequence; D030741:Carbonic Anhydrase IV / Q000235:genetics / Q000378:metabolism*; D018450:Disease Progression; D004721:Endoplasmic Reticulum / Q000378:metabolism; D005260:Female; D050956:HSP40 Heat-Shock Proteins / Q000172:deficiency / Q000378:metabolism*; D006801:Humans; D007668:Kidney / Q000201:enzymology* / Q000293:injuries / Q000473:pathology*; D008297:Male; D051379:Mice; D008822:Mice, Transgenic; D009154:Mutation; D017510:Protein Folding; D013312:Stress, Physiological; D015854:Up-Regulation
C500846:Dnajc3 protein, mouse; D050956:HSP40 Heat-Shock Proteins; D030741:Carbonic Anhydrase IV; C548247:CA4 protein, human