A non-functioning vitamin D receptor predisposes to leukaemoid reactions in mice.
Erben
Reinhold G
RG
Institute of Animal Physiology, Ludwig Maximilians University, Veterinärstrasse, Munich, Germany. Reinhold.Erben@vetmeduni.ac.at
Zeitz
Ute
U
Weber
Karin
K
Stierstorfer
Birgit
B
Wolf
Georg
G
Schmahl
Wolfgang
W
Balling
Rudi
R
Quintanilla-Martinez
Leticia
L
D000367:Age Factors; D000818:Animals; D001853:Bone Marrow / Q000378:metabolism / Q000473:pathology; D002454:Cell Differentiation / Q000235:genetics; D049109:Cell Proliferation; D005260:Female; D020022:Genetic Predisposition to Disease; D005838:Genotype; D015464:Leukemia, Myelogenous, Chronic, BCR-ABL Positive / Q000235:genetics* / Q000473:pathology / Q000503:physiopathology; D008297:Male; D051379:Mice; D008810:Mice, Inbred C57BL; D018345:Mice, Knockout; D022423:Myeloid Cells / Q000378:metabolism / Q000473:pathology; D009929:Organ Size; D018167:Receptors, Calcitriol / Q000235:genetics* / Q000502:physiology; D000685:Serum Amyloid A Protein / Q000378:metabolism; D013154:Spleen / Q000378:metabolism / Q000473:pathology; D013163:Splenomegaly / Q000235:genetics / Q000473:pathology
D018167:Receptors, Calcitriol; D000685:Serum Amyloid A Protein