Heamostatic and genetic predisposing factors for stroke in children with sickle cell anemia.
Mourad
Heba
H
Department of Clinical Pathology, Faculty of Medicine, Tanta University, Egypt.
Fadel
Wael
W
El Batch
Manal
M
Rowisha
Mohamed
M
D000755:Anemia, Sickle Cell / Q000097:blood* / Q000150:complications / Q000175:diagnosis; D000990:Antithrombin III; D015984:Causality; D002648:Child; D002675:Child, Preschool; D004534:Egypt / Q000453:epidemiology; D005260:Female; D005338:Fibrin Fibrinogen Degradation Products / Q000378:metabolism; D005344:Fibrinopeptide A / Q000378:metabolism; D005787:Gene Frequency / Q000235:genetics; D005838:Genotype; D006487:Hemostasis; D006801:Humans; D008297:Male; D010447:Peptide Hydrolases / Q000097:blood; D007703:Peptidyl-Dipeptidase A / Q000235:genetics*; D019408:Platelet Endothelial Cell Adhesion Molecule-1 / Q000097:blood; D011110:Polymorphism, Genetic / Q000235:genetics; D012307:Risk Factors; D020521:Stroke / Q000097:blood* / Q000453:epidemiology* / Q000209:etiology / Q000235:genetics*
D005338:Fibrin Fibrinogen Degradation Products; D019408:Platelet Endothelial Cell Adhesion Molecule-1; C046193:antithrombin III-protease complex; C036309:fibrin fragment D; D005344:Fibrinopeptide A; D000990:Antithrombin III; D010447:Peptide Hydrolases; C488034:ACE protein, human; D007703:Peptidyl-Dipeptidase A