Renal denervation reduces glomerular injury by suppressing NAD(P)H oxidase activity in Dahl salt-sensitive rats.
Nagasu
Hajime
H
Division of Nephrology, Department of Internal Medicine, Kawasaki Medical School, Kurashiki, Okayama, Japan.
Satoh
Minoru
M
Kuwabara
Atsunori
A
Yorimitsu
Daisuke
D
Sakuta
Takeo
T
Tomita
Naruya
N
Kashihara
Naoki
N
D000818:Animals; D000975:Antioxidants / Q000494:pharmacology; D001794:Blood Pressure / Q000187:drug effects; D015153:Blotting, Western; D002395:Catecholamines / Q000378:metabolism; D005923:Glomerulosclerosis, Focal Segmental / Q000201:enzymology* / Q000473:pathology / Q000517:prevention & control*; D006339:Heart Rate / Q000187:drug effects; D007668:Kidney / Q000187:drug effects / Q000294:innervation*; D008297:Male; D019255:NADPH Oxidases / Q000235:genetics / Q000378:metabolism*; D009478:Neuropeptide Y / Q000378:metabolism; D010084:Oxidation-Reduction; D012333:RNA, Messenger / Q000235:genetics; D051381:Rats; D020303:Rats, Inbred Dahl; D017207:Rats, Sprague-Dawley; D017382:Reactive Oxygen Species / Q000378:metabolism*; D020133:Reverse Transcriptase Polymerase Chain Reaction; D017673:Sodium Chloride, Dietary / Q000633:toxicity*
D000975:Antioxidants; D002395:Catecholamines; D009478:Neuropeptide Y; D012333:RNA, Messenger; D017382:Reactive Oxygen Species; D017673:Sodium Chloride, Dietary; D019255:NADPH Oxidases