Oxidative stress activates STAT1 in basilar arteries after subarachnoid hemorrhage.
Osuka
Koji
K
Department of Neurological Surgery, Aichi Medical University, Karimata Yazako, Nagakute, Aichi-gun, 480-1195, Japan.
Watanabe
Yasuo
Y
Usuda
Nobuteru
N
Atsuzawa
Kimie
K
Wakabayashi
Toshihiko
T
Takayasu
Masakazu
M
D000199:Actins / Q000378:metabolism; D000595:Amino Acid Sequence; D000818:Animals; D001488:Basilar Artery / Q000378:metabolism*; D015153:Blotting, Western; D004195:Disease Models, Animal; D016166:Free Radical Scavengers / Q000378:metabolism; D006861:Hydrogen Peroxide / Q000378:metabolism; D017665:Hydroxyl Radical / Q000378:metabolism; D007150:Immunohistochemistry; D015850:Interleukin-6 / Q000378:metabolism; D008297:Male; D018384:Oxidative Stress / Q000502:physiology*; D010766:Phosphorylation; D051381:Rats; D017207:Rats, Sprague-Dawley; D050794:STAT1 Transcription Factor / Q000235:genetics / Q000378:metabolism*; D050796:STAT3 Transcription Factor / Q000378:metabolism; D013345:Subarachnoid Hemorrhage / Q000378:metabolism*; D013997:Time Factors
D000199:Actins; D016166:Free Radical Scavengers; D015850:Interleukin-6; D050794:STAT1 Transcription Factor; D050796:STAT3 Transcription Factor; C494081:Stat1 protein, rat; C494088:Stat3 protein, rat; D017665:Hydroxyl Radical; D006861:Hydrogen Peroxide