TAX1BP1 and A20 inhibit antiviral signaling by targeting TBK1-IKKi kinases.
Parvatiyar
Kislay
K
Department of Microbiology and Immunology, University of Miami Miller School of Medicine, Miami, Florida 33136.
Barber
Glen N
GN
Department of Microbiology and Immunology, University of Miami Miller School of Medicine, Miami, Florida 33136; Department of Medicine, Sylvester Comprehensive Cancer Center, University of Miami Miller School of Medicine, Miami, Florida 33136.
Harhaj
Edward W
EW
Department of Microbiology and Immunology, University of Miami Miller School of Medicine, Miami, Florida 33136. Electronic address: eharhaj@med.miami.edu.
D000818:Animals; D002460:Cell Line; D004268:DNA-Binding Proteins; D004797:Enzyme-Linked Immunosorbent Assay; D006801:Humans; D047908:Intracellular Signaling Peptides and Proteins / Q000502:physiology*; D051379:Mice; D009363:Neoplasm Proteins / Q000502:physiology*; D009687:Nuclear Proteins / Q000502:physiology*; D011494:Protein Kinases / Q000378:metabolism*; D017346:Protein Serine-Threonine Kinases / Q000378:metabolism*; D015398:Signal Transduction; D000072598:Tumor Necrosis Factor alpha-Induced Protein 3; D018116:Vesiculovirus / Q000472:pathogenicity*
D004268:DNA-Binding Proteins; D047908:Intracellular Signaling Peptides and Proteins; D009363:Neoplasm Proteins; D009687:Nuclear Proteins; C120635:TAX1BP1 protein, human; C523855:TAX1BP1 protein, mouse; D011494:Protein Kinases; D017346:Protein Serine-Threonine Kinases; C403191:TBK1 protein, human; C076529:TNFAIP3 protein, human; D000072598:Tumor Necrosis Factor alpha-Induced Protein 3