Effect of genetic deletion of the vanilloid receptor TRPV1 on the expression of Substance P in sensory neurons of mice with adjuvant-induced arthritis.
Willcockson
Helen H
HH
Department of Cell and Developmental Biology, University of North Carolina, Chapel Hill, NC 27599, USA. hhw@med.unc.edu
Chen
Yong
Y
Han
Ji Eun
JE
Valtschanoff
Juli G
JG
D000818:Animals; D001169:Arthritis, Experimental / Q000378:metabolism*; D002452:Cell Count; D048429:Cell Size; D003627:Data Interpretation, Statistical; D005455:Fluorescent Antibody Technique; D005727:Ganglia, Spinal / Q000166:cytology / Q000378:metabolism; D007150:Immunohistochemistry; D008297:Male; D051379:Mice; D008810:Mice, Inbred C57BL; D018345:Mice, Knockout; D009413:Nerve Fibers, Myelinated / Q000502:physiology; D011984:Sensory Receptor Cells / Q000378:metabolism*; D013373:Substance P / Q000096:biosynthesis*; D050916:TRPV Cation Channels / Q000235:genetics* / Q000502:physiology*
D050916:TRPV Cation Channels; C493921:TRPV1 protein, mouse; D013373:Substance P