Abnormal mitochondrial function during ischemia reperfusion provides targets for pharmacological therapy.
García-Rivas
Gerardo J
GJ
Escuela de Medicina CITES, Tecnológico de Monterrey, Instituto de Cardiología y Medicina Vascular, Tecnológico de Monterrey, Monterrey, México.
Torre-Amione
Guillermo
G
D000255:Adenosine Triphosphate / Q000378:metabolism; D000818:Animals; D017209:Apoptosis; D001145:Arrhythmias, Cardiac / Q000378:metabolism / Q000473:pathology / Q000517:prevention & control*; D002118:Calcium / Q000378:metabolism; D002317:Cardiovascular Agents / Q000627:therapeutic use*; D006801:Humans; D053078:Membrane Potential, Mitochondrial; D008929:Mitochondria, Heart / Q000187:drug effects* / Q000378:metabolism / Q000473:pathology; D033681:Mitochondrial Membrane Transport Proteins / Q000378:metabolism; D000083162:Mitochondrial Permeability Transition Pore; D015428:Myocardial Reperfusion Injury / Q000378:metabolism / Q000473:pathology / Q000517:prevention & control*; D017382:Reactive Oxygen Species / Q000378:metabolism; D015398:Signal Transduction; D013997:Time Factors
D002317:Cardiovascular Agents; D033681:Mitochondrial Membrane Transport Proteins; D000083162:Mitochondrial Permeability Transition Pore; D017382:Reactive Oxygen Species; D000255:Adenosine Triphosphate; D002118:Calcium