Loss of SNAP29 impairs endocytic recycling and cell motility.
Rapaport
Debora
D
Department of Cell Research and Immunology, Tel Aviv University, Ramat Aviv, Israel.
Lugassy
Yevgenia
Y
Sprecher
Eli
E
Horowitz
Mia
M
D002460:Cell Line; D002465:Cell Movement; D002772:Cholera Toxin / Q000378:metabolism; D004705:Endocytosis; D005089:Exocytosis; D005347:Fibroblasts / Q000378:metabolism*; D006056:Golgi Apparatus / Q000378:metabolism; D006801:Humans; D019012:Integrin beta1 / Q000378:metabolism; D008954:Models, Biological; D010766:Phosphorylation; D017434:Protein Structure, Tertiary; D050766:Qb-SNARE Proteins / Q000378:metabolism*; D050767:Qc-SNARE Proteins / Q000378:metabolism*; D014168:Transferrin / Q000737:chemistry; D014945:Wound Healing
D019012:Integrin beta1; D050766:Qb-SNARE Proteins; D050767:Qc-SNARE Proteins; C432420:SNAP29 protein, human; D014168:Transferrin; D002772:Cholera Toxin