Acute hypoxia promote the phosphorylation of tau via ERK pathway.
Fang
Hui
H
Key Laboratory of Brain Function and Diseases, School of Life Science, University of Science and Technology of China, Chinese Academy of Sciences, Hefei 230027, Anhui, China.
Zhang
Li-Feng
LF
Key Laboratory of Brain Function and Diseases, School of Life Science, University of Science and Technology of China, Chinese Academy of Sciences, Hefei 230027, Anhui, China.
Meng
Fan-Tao
FT
Key Laboratory of Brain Function and Diseases, School of Life Science, University of Science and Technology of China, Chinese Academy of Sciences, Hefei 230027, Anhui, China.
Du
Xin
X
Key Laboratory of Brain Function and Diseases, School of Life Science, University of Science and Technology of China, Chinese Academy of Sciences, Hefei 230027, Anhui, China.
Zhou
Jiang-Ning
JN
Key Laboratory of Brain Function and Diseases, School of Life Science, University of Science and Technology of China, Chinese Academy of Sciences, Hefei 230027, Anhui, China. Electronic address: jnzhou@ustc.edu.cn.
D000818:Animals; D015687:Cell Hypoxia / Q000502:physiology*; D002460:Cell Line; D049109:Cell Proliferation; D048049:Extracellular Signal-Regulated MAP Kinases / Q000378:metabolism*; D006624:Hippocampus / Q000378:metabolism* / Q000503:physiopathology; D006801:Humans; D000860:Hypoxia / Q000503:physiopathology*; D015151:Immunoblotting; D051379:Mice; D008810:Mice, Inbred C57BL; D010766:Phosphorylation; D015398:Signal Transduction / Q000502:physiology*; D016875:tau Proteins / Q000378:metabolism*
D016875:tau Proteins; D048049:Extracellular Signal-Regulated MAP Kinases