Role of excessive inflammatory response to Stenotrophomonas maltophilia lung infection in DBA/2 mice and implications for cystic fibrosis.
Di Bonaventura
Giovanni
G
Department of Biomedical Sciences, G d'Annunzio University of Chieti-Pescara, Chieti, Italy. gdibonaventura@unich.it
Pompilio
Arianna
A
Zappacosta
Roberta
R
Petrucci
Francesca
F
Fiscarelli
Ersilia
E
Rossi
Cosmo
C
Piccolomini
Raffaele
R
D000818:Animals; D001835:Body Weight; D015169:Colony Count, Microbial; D003550:Cystic Fibrosis / Q000276:immunology / Q000382:microbiology* / Q000401:mortality / Q000473:pathology*; D016207:Cytokines / Q000378:metabolism; D005260:Female; D007249:Inflammation; D008168:Lung / Q000382:microbiology* / Q000473:pathology*; D008297:Male; D051379:Mice; D008811:Mice, Inbred DBA; D009504:Neutrophils / Q000276:immunology; D020615:Stenotrophomonas maltophilia / Q000276:immunology* / Q000472:pathogenicity*; D016019:Survival Analysis
D016207:Cytokines