Neurochemical alterations in spinocerebellar ataxia type 1 and their correlations with clinical status.
Oz
Gülin
G
Center for MR Research, Department of Radiology, Medical School, University of Minnesota, Minneapolis, Minnesota 55455, USA. gulin@cmrr.umn.edu
Hutter
Diane
D
Tkác
Ivan
I
Clark
H Brent
HB
Gross
Myron D
MD
Jiang
Hong
H
Eberly
Lynn E
LE
Bushara
Khalaf O
KO
Gomez
Christopher M
CM
D000704:Analysis of Variance; D001224:Aspartic Acid / Q000031:analogs & derivatives; D001923:Brain Chemistry; D016022:Case-Control Studies; D005260:Female; D007839:Functional Laterality; D005904:Glial Fibrillary Acidic Protein / Q000134:cerebrospinal fluid; D006801:Humans; D007294:Inositol; D028421:Isoprostanes / Q000134:cerebrospinal fluid; D008279:Magnetic Resonance Imaging / Q000379:methods; D009682:Magnetic Resonance Spectroscopy / Q000379:methods; D008297:Male; D008875:Middle Aged; D010725:Phosphocreatine; D011522:Protons; D012720:Severity of Illness Index; D020754:Spinocerebellar Ataxias / Q000378:metabolism* / Q000503:physiopathology*; D013223:Statistics as Topic
D005904:Glial Fibrillary Acidic Protein; D028421:Isoprostanes; D011522:Protons; D010725:Phosphocreatine; D001224:Aspartic Acid; D007294:Inositol; C000179:N-acetylaspartate