Truncated beta-amyloid peptide channels provide an alternative mechanism for Alzheimer's Disease and Down syndrome.
Jang
Hyunbum
H
Center for Cancer Research Nanobiology Program, SAIC-Frederick, Inc, National Cancer Institute, Frederick, MD 21702, USA.
Arce
Fernando Teran
FT
Ramachandran
Srinivasan
S
Capone
Ricardo
R
Azimova
Rushana
R
Kagan
Bruce L
BL
Nussinov
Ruth
R
Lal
Ratnesh
R
D000544:Alzheimer Disease / Q000378:metabolism* / Q000473:pathology; D016229:Amyloid beta-Peptides / Q000378:metabolism* / Q000648:ultrastructure; D002118:Calcium / Q000378:metabolism; D016923:Cell Death; D004314:Down Syndrome / Q000378:metabolism* / Q000473:pathology; D006801:Humans; D018625:Microscopy, Atomic Force; D008958:Models, Molecular; D010446:Peptide Fragments / Q000378:metabolism* / Q000648:ultrastructure; D017434:Protein Structure, Tertiary
D016229:Amyloid beta-Peptides; D010446:Peptide Fragments; C549047:amyloid beta-peptide (9-42); C000605559:amyloid beta-protein (11-42); C086839:amyloid beta-protein (17-42); D002118:Calcium