Matrix metalloproteinase-9 activity increased by two different types of epileptic seizures that do not induce neuronal death: a possible role in homeostatic synaptic plasticity.
Takács
Eszter
E
Laboratory of Proteomics, Institute of Biology, Eötvös Loránd University, Budapest, Hungary. neuroproteome@gmail.com
Nyilas
Rita
R
Szepesi
Zsuzsanna
Z
Baracskay
Péter
P
Karlsen
Bente
B
Røsvold
Tina
T
Bjørkum
Alvhild A
AA
Czurkó
András
A
Kovács
Zsolt
Z
Kékesi
Adrienna K
AK
Juhász
Gábor
G
D015761:4-Aminopyridine; D000818:Animals; D001522:Behavior, Animal; D016923:Cell Death; D004195:Disease Models, Animal; D004569:Electroencephalography; D004827:Epilepsy / Q000139:chemically induced / Q000201:enzymology* / Q000503:physiopathology; D005625:Frontal Lobe / Q000201:enzymology; D006706:Homeostasis; D008297:Male; D020778:Matrix Metalloproteinase 2 / Q000378:metabolism; D020780:Matrix Metalloproteinase 9 / Q000378:metabolism*; D009473:Neuronal Plasticity / Q000502:physiology*; D009474:Neurons / Q000502:physiology*; D010296:Parietal Lobe / Q000201:enzymology; D026902:Potassium Channel Blockers; D051381:Rats; D017207:Rats, Sprague-Dawley; D017208:Rats, Wistar; D013569:Synapses / Q000502:physiology*; D013788:Thalamus / Q000201:enzymology
D026902:Potassium Channel Blockers; D015761:4-Aminopyridine; D020778:Matrix Metalloproteinase 2; D020780:Matrix Metalloproteinase 9