Helicobacter pylori CagL activates ADAM17 to induce repression of the gastric H, K-ATPase alpha subunit.
Saha
Arindam
A
Department of Medicine, University of California, San Diego, San Diego, California, USA.
Backert
Steffen
S
Hammond
Charles E
CE
Gooz
Monika
M
Smolka
Adam J
AJ
D051722:ADAM Proteins / Q000502:physiology*; D000072198:ADAM17 Protein; D004794:Enzyme Repression; D016480:Helicobacter pylori / Q000502:physiology*; D066257:Heparin-binding EGF-like Growth Factor; D006801:Humans; D019012:Integrin beta1 / Q000378:metabolism; D036341:Intercellular Signaling Peptides and Proteins / Q000378:metabolism; D009842:Oligopeptides / Q000502:physiology; D011401:Promoter Regions, Genetic; D000254:Sodium-Potassium-Exchanging ATPase / Q000096:biosynthesis / Q000235:genetics*; D013270:Stomach / Q000201:enzymology*
C587883:HBEGF protein, human; D066257:Heparin-binding EGF-like Growth Factor; D019012:Integrin beta1; D036341:Intercellular Signaling Peptides and Proteins; D009842:Oligopeptides; C047981:arginyl-glycyl-aspartic acid; D051722:ADAM Proteins; D000072198:ADAM17 Protein; C000606462:ADAM17 protein, human; C499076:ATP1A1 protein, human; D000254:Sodium-Potassium-Exchanging ATPase