The LTB4-BLT1 axis mediates neutrophil infiltration and secondary injury in experimental spinal cord injury.
Saiwai
Hirokazu
H
Department of Research Superstar Program Stem Cell Unit, Graduate School of Medical Sciences, Kyushu University, Fukuoka, Japan.
Ohkawa
Yasuyuki
Y
Yamada
Hisakata
H
Kumamaru
Hiromi
H
Harada
Akihito
A
Okano
Hideyuki
H
Yokomizo
Takehiko
T
Iwamoto
Yukihide
Y
Okada
Seiji
S
D000818:Animals; D017209:Apoptosis; D053222:Fas Ligand Protein / Q000096:biosynthesis; D005260:Female; D005434:Flow Cytometry / Q000379:methods; D005786:Gene Expression Regulation; D053583:Interleukin-1beta / Q000096:biosynthesis; D015850:Interleukin-6 / Q000096:biosynthesis; D007975:Leukotriene B4 / Q000378:metabolism / Q000502:physiology*; D051379:Mice; D008810:Mice, Inbred C57BL; D009504:Neutrophils / Q000378:metabolism*; D018102:Receptors, Leukotriene B4 / Q000378:metabolism / Q000502:physiology*; D013116:Spinal Cord / Q000473:pathology*; D013119:Spinal Cord Injuries / Q000473:pathology*; D014409:Tumor Necrosis Factor-alpha / Q000096:biosynthesis
D053222:Fas Ligand Protein; D053583:Interleukin-1beta; D015850:Interleukin-6; C503704:Ltb4r1 protein, mouse; D018102:Receptors, Leukotriene B4; D014409:Tumor Necrosis Factor-alpha; D007975:Leukotriene B4