Th17 cells promote autoimmune anti-myeloperoxidase glomerulonephritis.
Gan
Poh-Yi
PY
Centre for Inflammatory Diseases, Monash University Department of Medicine, Monash Medical Centre, 246 Clayton Road, Clayton, VIC 3168, Australia.
Steinmetz
Oliver M
OM
Tan
Diana S Y
DS
O'Sullivan
Kim M
KM
Ooi
Joshua D
JD
Iwakura
Yoichiro
Y
Kitching
A Richard
AR
Holdsworth
Stephen R
SR
D000818:Animals; D000906:Antibodies / Q000378:metabolism*; D019268:Antibodies, Antineutrophil Cytoplasmic / Q000378:metabolism; D001327:Autoimmune Diseases / Q000209:etiology / Q000378:metabolism* / Q000473:pathology; D004195:Disease Models, Animal; D005921:Glomerulonephritis / Q000209:etiology / Q000378:metabolism* / Q000473:pathology; D020381:Interleukin-17 / Q000235:genetics / Q000378:metabolism*; D007668:Kidney / Q000187:drug effects / Q000378:metabolism; D008264:Macrophages / Q000378:metabolism / Q000473:pathology; D008297:Male; D051379:Mice; D008810:Mice, Inbred C57BL; D018345:Mice, Knockout; D009504:Neutrophils / Q000378:metabolism / Q000473:pathology; D009195:Peroxidase / Q000276:immunology* / Q000494:pharmacology; D016176:T-Lymphocyte Subsets / Q000378:metabolism* / Q000473:pathology
D000906:Antibodies; D019268:Antibodies, Antineutrophil Cytoplasmic; D020381:Interleukin-17; D009195:Peroxidase