title: The CACNA1C risk variant for bipolar disorder influences limbic activity.
authors.forename: M
authors.initials: M
authors.lastname: Wessa
authors.forename: J
authors.initials: J
authors.lastname: Linke
authors.forename: S H
authors.initials: SH
authors.lastname: Witt
authors.forename: V
authors.initials: V
authors.lastname: Nieratschker
authors.forename: C
authors.initials: C
authors.lastname: Esslinger
authors.forename: P
authors.initials: P
authors.lastname: Kirsch
authors.forename: O
authors.initials: O
authors.lastname: Grimm
authors.forename: M G
authors.initials: MG
authors.lastname: Hennerici
authors.forename: A
authors.initials: A
authors.lastname: Gass
authors.forename: A V
authors.initials: AV
authors.lastname: King
authors.forename: M
authors.initials: M
authors.lastname: Rietschel
mesh_terms: D000328:Adult; D001714:Bipolar Disorder / Q000235:genetics* / Q000378:metabolism; D001931:Brain Mapping; D020746:Calcium Channels, L-Type / Q000235:genetics / Q000378:metabolism*; D058068:Endophenotypes / Q000378:metabolism*; D005260:Female; D018628:Gene Dosage / Q000502:physiology; D006801:Humans; D008032:Limbic System / Q000502:physiology*; D008279:Magnetic Resonance Imaging; D008297:Male; D012016:Reference Values; D055815:Young Adult
chemical_list: C079146:CACNA1C protein, human; D020746:Calcium Channels, L-Type