The role of TREK-1 in cerebrovascular regulation remains elusive: focus on "Cerebrovascular responses in mice deficient in the potassium channel, TREK-1".
Muller-Delp
J M
JM
D000200:Action Potentials; D000818:Animals; D001011:Aorta / Q000378:metabolism; D016718:Arachidonic Acid / Q000378:metabolism; D001488:Basilar Artery / Q000378:metabolism*; D002560:Cerebrovascular Circulation; D006801:Humans; D051036:Large-Conductance Calcium-Activated Potassium Channels / Q000378:metabolism; D051379:Mice; D020768:Middle Cerebral Artery / Q000378:metabolism*; D009131:Muscle, Smooth, Vascular / Q000378:metabolism; D032389:Myocytes, Smooth Muscle / Q000378:metabolism; D009569:Nitric Oxide / Q000378:metabolism; D011188:Potassium / Q000378:metabolism*; D024683:Potassium Channels, Tandem Pore Domain / Q000172:deficiency* / Q000235:genetics; D011451:Prostaglandin-Endoperoxide Synthases / Q000378:metabolism; D014661:Vasoconstriction; D014664:Vasodilation; D017962:alpha-Linolenic Acid / Q000378:metabolism
D051036:Large-Conductance Calcium-Activated Potassium Channels; D024683:Potassium Channels, Tandem Pore Domain; C103742:potassium channel protein TREK-1; D017962:alpha-Linolenic Acid; D016718:Arachidonic Acid; D009569:Nitric Oxide; D011451:Prostaglandin-Endoperoxide Synthases; D011188:Potassium