title: [Problem of end effector of delayed ischemic preconditioning of the heart].
abstract: Analysis of published data on the problem of end effector of delayed ischemic preconditioning of the heart presented evidence that NO-synthase, superoxide dismutase, cyclooxygenase-2, heme oxygenase-1 are the main pretenders to the role of end effector of delayed ischemic preconditioning.
authors.forename: Iu B
authors.initials: IuB
authors.lastname: Lishmanov
authors.forename: L N
authors.initials: LN
authors.lastname: Maslov
authors.forename: T
authors.initials: T
authors.lastname: Krieg
authors.forename: I G
authors.initials: IG
authors.lastname: Khaliulin
mesh_terms: D000818:Animals; D051546:Cyclooxygenase 2 / Q000378:metabolism*; D051547:Heme Oxygenase-1 / Q000378:metabolism*; D006801:Humans; D019157:Ischemic Preconditioning, Myocardial; D009124:Muscle Proteins / Q000378:metabolism*; D009206:Myocardium / Q000201:enzymology*; D019001:Nitric Oxide Synthase / Q000378:metabolism*; D013482:Superoxide Dismutase / Q000378:metabolism*
chemical_list: D009124:Muscle Proteins; D019001:Nitric Oxide Synthase; D051547:Heme Oxygenase-1; D051546:Cyclooxygenase 2; C496540:PTGS2 protein, human; D013482:Superoxide Dismutase