Neuropsychology of acute stroke.
Neuropsychology includes both the psychiatric manifestations of neurological illness (primary brain-based disorders) and neurobiology of "idiopathic" psychiatric disorders. Neurological primary brain disorders provoke broad spectrum of brain pathophysiology that cause deficit sin human behaviour, and the magnitude of neurobehavioral-related problems is a world wide health concern. Speech disorders of aphasic type, unilateral neglect, anosognosia (deficit disorders), delirium and mood disorders (productive disorders) in urgent neurology, first of all in acute phase of stroke are more frequent disorders then it verified in routine exam, not only in the developed and large neurological departments. Aphasia is common consequence of left hemispheric lesion and most common neuropsychological consequence of stroke, with prevalence of one third of all stroke patients in acute phase although exist reports on greater frequency. Unilateral neglect is a disorder that mostly effects the patient after the lesion of the right hemisphere, mostly caused by a cerebrovascular insult (infarct or haemorrhage affecting a large area - up to two thirds of the right hemisphere), and in general the left-side neglect is the most widespread neuropsychological deficit after the lesion of the right cerebral hemisphere. Reports on the incidence of visual neglect vary and they range from 13 to 85%. Anosognosia is on the second place as neuropsychological syndrome of stroke in right hemisphere, characterized by the denial of the motor, visual or cognitive deficit. This syndrome, defined as denial of hemiparesis or hemianopsia, is a common disorder verified in 17-28% of all patents with acute brain stoke. There are different reports on frequency of delirium in acute stroke, from 24 to 48%, and it is more frequent in hemorrhagic then ischemic stoke. Post stroke depression (PSD) is one of the more frequent consequences on the stroke, and the prevalence of PSD has ranged from 5 to 63% of patients in several cross-sectional studies, peaking three to six months after a stroke.
Sinanović
Osman
O
Department of Neurology, University Clinical Center Tuzla, Medical Faculty University of Tuzla, 75000 Tuzla, Bosnia and Herzegovina. osman.sinanovic@ukctuzla.ba
D000377:Agnosia / Q000175:diagnosis / Q000503:physiopathology / Q000523:psychology; D001037:Aphasia / Q000175:diagnosis / Q000503:physiopathology / Q000523:psychology; D001364:Awareness / Q000502:physiology; D002544:Cerebral Infarction / Q000175:diagnosis / Q000503:physiopathology / Q000523:psychology*; D003693:Delirium / Q000175:diagnosis / Q000503:physiopathology / Q000523:psychology; D003717:Denial, Psychological; D003866:Depressive Disorder / Q000175:diagnosis / Q000503:physiopathology / Q000523:psychology; D004292:Dominance, Cerebral / Q000502:physiology; D006423:Hemianopsia / Q000175:diagnosis / Q000503:physiopathology / Q000523:psychology; D006429:Hemiplegia / Q000175:diagnosis / Q000503:physiopathology / Q000523:psychology; D006801:Humans; D019965:Neurocognitive Disorders / Q000175:diagnosis / Q000503:physiopathology / Q000523:psychology*; D010468:Perceptual Disorders / Q000175:diagnosis / Q000503:physiopathology / Q000523:psychology